Inactivation of the somatosensory cortex prevents paroxysmal oscillations in cortical and related thalamic neurons in a genetic model of absence epilepsy.
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Anterior Cingulate epilepsy: mechanisms and modulationCaV 2.1 ablation in cortical interneurons selectively impairs fast-spiking basket cells and causes generalized seizures.Spatiotemporal dynamics of optogenetically induced and spontaneous seizure transitions in primary generalized epilepsy.Persistence of cortical sensory processing during absence seizures in human and an animal model: evidence from EEG and intracellular recordings.Reduced local input to fast-spiking interneurons in the somatosensory cortex in the GABAA γ2 R43Q mouse model of absence epilepsy.Aberrant GABA(A) receptor-mediated inhibition in cortico-thalamic networks of succinic semialdehyde dehydrogenase deficient mice.Rhythmic neuronal activity in S2 somatosensory and insular cortices contribute to the initiation of absence-related spike-and-wave discharges.Mapping epileptic activity: sources or networks for the clinicians?Effects of childhood absence epilepsy on associations between regional cortical morphometry and aging and cognitive abilitiesSubthalamic nucleus high-frequency stimulation restores altered electrophysiological properties of cortical neurons in parkinsonian ratThalamocortical neurons display suppressed burst-firing due to an enhanced Ih current in a genetic model of absence epilepsyUnilateral and Bilateral Cortical Resection: Effects on Spike-Wave Discharges in a Genetic Absence Epilepsy Model.Complexity of Multi-Channel Electroencephalogram Signal Analysis in Childhood Absence Epilepsy.Ethosuximide Affects Paired-Pulse Facilitation in Somatosensory Cortex of WAG\Rij Rats as a Model of Absence Seizure.Striatal GABAergic and cortical glutamatergic neurons mediate contrasting effects of cannabinoids on cortical network synchrony.Neurochemical and behavioral features in genetic absence epilepsy and in acutely induced absence seizures.Cortical and subcortical brain alterations in Juvenile Absence Epilepsy.Primary motor and sensory cortical areas communicate via spatiotemporally coordinated networks at multiple frequenciesDynamics of networks during absence seizure's on- and offset in rodents and man.Epileptic neuronal networks: methods of identification and clinical relevanceDynamics of sensorimotor cortex activation during absence and myoclonic seizures in a mouse model of juvenile myoclonic epilepsy.The hippocampus participates in a pharmacological rat model of absence seizuresPotentiation of mGlu5 receptors with the novel enhancer, VU0360172, reduces spontaneous absence seizures in WAG/Rij ratsTransplantation of GABAergic Interneurons into the Neonatal Primary Visual Cortex Reduces Absence Seizures in Stargazer Mice.Bidirectional Control of Generalized Epilepsy Networks via Rapid Real-Time Switching of Firing Mode.Seizure expression, behavior, and brain morphology differences in colonies of Genetic Absence Epilepsy Rats from Strasbourg.Macroscopic and microscopic spectral properties of brain networks during local and global synchronization.Spatiotemporal organization and thalamic modulation of seizures in the mouse medial thalamic-anterior cingulate slice.Developmental changes in Notch1 and NLE1 expression in a genetic model of absence epilepsy.Integrative properties and transfer function of cortical neurons initiating absence seizures in a rat genetic model.Focal administration of neuropeptide Y into the S2 somatosensory cortex maximally suppresses absence seizures in a genetic rat model.Effect of intracranial administration of ethosuximide in rats with spontaneous or pentylenetetrazol-induced spike-wave discharges.Building Up Absence Seizures in the Somatosensory Cortex: From Network to Cellular Epileptogenic Processes.Closed-loop seizure control with very high frequency electrical stimulation at seizure onset in the GAERS model of absence epilepsy.CaV 3.2 drives sustained burst-firing, which is critical for absence seizure propagation in reticular thalamic neurons.Disrupted Coupling Between the Spontaneous Fluctuation and Functional Connectivity in Idiopathic Generalized EpilepsyImaging fast electrical activity in the brain during ictal epileptiform discharges with electrical impedance tomography
P2860
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P2860
Inactivation of the somatosensory cortex prevents paroxysmal oscillations in cortical and related thalamic neurons in a genetic model of absence epilepsy.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Inactivation of the somatosens ...... tic model of absence epilepsy.
@en
Inactivation of the somatosens ...... tic model of absence epilepsy.
@nl
type
label
Inactivation of the somatosens ...... tic model of absence epilepsy.
@en
Inactivation of the somatosens ...... tic model of absence epilepsy.
@nl
prefLabel
Inactivation of the somatosens ...... tic model of absence epilepsy.
@en
Inactivation of the somatosens ...... tic model of absence epilepsy.
@nl
P2860
P356
P1433
P1476
Inactivation of the somatosens ...... tic model of absence epilepsy.
@en
P2093
Stéphane Charpier
Séverine Mahon
P2860
P304
P356
10.1093/CERCOR/BHN237
P577
2009-03-10T00:00:00Z