Relationship between Na+-Ca2+-exchanger protein levels and diastolic function of failing human myocardium.
about
Transcriptional pathways and potential therapeutic targets in the regulation of Ncx1 expression in cardiac hypertrophy and failureAbnormal Ca(2+) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balanceThe ryanodine receptor leak: how a tattered receptor plunges the failing heart into crisisRegulation of intracellular Na(+) in health and disease: pathophysiological mechanisms and implications for treatmentCalcium mishandling in diastolic dysfunction: mechanisms and potential therapiesBIN1 localizes the L-type calcium channel to cardiac T-tubulesIncreased Na⁺/Ca²⁺ exchanger expression/activity constitutes a point of inflection in the progression to heart failure of hypertensive ratsCardiovascular risks associated with low dose ionizing particle radiationMechanistic insight into the functional and toxic effects of Strophanthidin in the failing human myocardiumModelling sarcomeric cardiomyopathies in the dish: from human heart samples to iPSC cardiomyocytesImprovement in survival and cardiac metabolism after gene transfer of sarcoplasmic reticulum Ca(2+)-ATPase in a rat model of heart failureMaturing human pluripotent stem cell-derived cardiomyocytes in human engineered cardiac tissuesHistone deacetylases facilitate sodium/calcium exchanger up-regulation in adult cardiomyocytesEarly-onset growth hormone deficiency results in diastolic dysfunction in adult-life and is prevented by growth hormone supplementationRegulation of Ncx1 expression. Identification of regulatory elements mediating cardiac-specific expression and up-regulation.Aerobic interval training partly reverse contractile dysfunction and impaired Ca2+ handling in atrial myocytes from rats with post infarction heart failureOverexpression of the Na(+)/Ca(2+) exchanger and inhibition of the sarcoplasmic reticulum Ca(2+)-ATPase in ventricular myocytes from transgenic mice.Low-dose ramipril treatment improves relaxation and calcium cycling after established cardiac hypertrophy.In vivo regulation of Na/Ca exchanger expression by adrenergic effectors.Impact of detubulation on force and kinetics of cardiac muscle contraction.Influence of a constitutive increase in myofilament Ca(2+)-sensitivity on Ca(2+)-fluxes and contraction of mouse heart ventricular myocytesBaroreflex deficiency induces additional impairment of vagal tone, diastolic function and calcium handling proteins after myocardial infarction.Rescue of familial cardiomyopathies by modifications at the level of sarcomere and Ca2+ fluxes.Compensatory and decompensatory alterations in cardiomyocyte Ca2+ dynamics in hearts with diastolic dysfunction following aortic banding.Late sodium current contributes to diastolic cell Ca2+ accumulation in chronic heart failure.Targeting phospholamban by gene transfer in human heart failure.Decoding calcium signals involved in cardiac growth and function.Mechanisms of calcium buffering in adrenergic neurones and effects of ageing: testing the limits of homeostasis.Gene delivery of sarcoplasmic reticulum calcium ATPase inhibits ventricular remodeling in ischemic mitral regurgitationGenomics and the pathophysiology of heart failure.Neonatal gene transfer of Serca2a delays onset of hypertrophic remodeling and improves function in familial hypertrophic cardiomyopathy.Defects in T-tubular electrical activity underlie local alterations of calcium release in heart failureThe Na+/Ca2+ exchanger-1 mediates left ventricular dysfunction in mice with chronic intermittent hypoxia.Calcium dynamics in the ventricular myocytes of SERCA2 knockout mice: A modeling studyCardiac-specific elevations in thyroid hormone enhance contractility and prevent pressure overload-induced cardiac dysfunction.Novel aspects of excitation-contraction coupling in heart failure.Full-length cardiac Na+/Ca2+ exchanger 1 protein is not phosphorylated by protein kinase A.Targeting calcium cycling proteins in heart failure through gene transfer.Altered myocardial calcium cycling and energetics in heart failure--a rational approach for disease treatment.Triple threat: the Na+/Ca2+ exchanger in the pathophysiology of cardiac arrhythmia, ischemia and heart failure.
P2860
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P2860
Relationship between Na+-Ca2+-exchanger protein levels and diastolic function of failing human myocardium.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh-hant
name
Relationship between Na+-Ca2+- ...... n of failing human myocardium.
@en
Relationship between Na+-Ca2+- ...... n of failing human myocardium.
@nl
type
label
Relationship between Na+-Ca2+- ...... n of failing human myocardium.
@en
Relationship between Na+-Ca2+- ...... n of failing human myocardium.
@nl
prefLabel
Relationship between Na+-Ca2+- ...... n of failing human myocardium.
@en
Relationship between Na+-Ca2+- ...... n of failing human myocardium.
@nl
P2093
P356
P1433
P1476
Relationship between Na+-Ca2+- ...... n of failing human myocardium.
@en
P2093
Hasenfuss G
Lehnart SE
Schillinger W
P304
P356
10.1161/01.CIR.99.5.641
P407
P577
1999-02-01T00:00:00Z