Targeting ROR1 inhibits the self-renewal and invasive ability of glioblastoma stem cells.
about
Distinct Patterns of Stromal and Tumor Expression of ROR1 and ROR2 in Histological Subtypes of Epithelial Ovarian Cancer.The ginsenoside metabolite compound K inhibits growth, migration and stemness of glioblastoma cells.An ROR1 bi-specific T-cell engager provides effective targeting and cytotoxicity against a range of solid tumors.In-Silico Drug discovery approach targeting receptor tyrosine kinase-like orphan receptor 1 for cancer treatment.
P2860
Targeting ROR1 inhibits the self-renewal and invasive ability of glioblastoma stem cells.
description
2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年學術文章
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2016年學術文章
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2016年學術文章
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name
Targeting ROR1 inhibits the self-renewal and invasive ability of glioblastoma stem cells.
@en
Targeting ROR1 inhibits the self-renewal and invasive ability of glioblastoma stem cells.
@nl
type
label
Targeting ROR1 inhibits the self-renewal and invasive ability of glioblastoma stem cells.
@en
Targeting ROR1 inhibits the self-renewal and invasive ability of glioblastoma stem cells.
@nl
prefLabel
Targeting ROR1 inhibits the self-renewal and invasive ability of glioblastoma stem cells.
@en
Targeting ROR1 inhibits the self-renewal and invasive ability of glioblastoma stem cells.
@nl
P2093
P2860
P356
P1476
Targeting ROR1 inhibits the self-renewal and invasive ability of glioblastoma stem cells.
@en
P2093
Chan-Wha Kim
Eun-Hwa Jung
Gi-Yeon Han
Han-Na Lee
Min-Jung Kim
P2860
P304
P356
10.1002/CBF.3172
P577
2016-02-28T00:00:00Z