Selective contribution of interleukin-6 and leptin to brain inflammatory signals induced by systemic LPS injection in mice.
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Angiopoietin-like protein 4: health effects, modulating agents and structure-function relationshipsEffectiveness of conservative interventions for sickness and pain behaviors induced by a high repetition high force upper extremity task.Unmyelinated nerve fibers in the human dental pulp express markers for myelinated fibers and show sodium channel accumulations.Molecular and cellular neuroinflammatory status of mouse brain after systemic lipopolysaccharide challenge: importance of CCR2/CCL2 signaling.Neonatal programming of innate immune functionAtypical protein kinase C activity in the hypothalamus is required for lipopolysaccharide-mediated sickness responses.Prebiotics to manage the microbial control of energy homeostasis.The immune theory of psychiatric diseases: a key role for activated microglia and circulating monocytes.Role of Angptl4 in vascular permeability and inflammation.In-depth conversation: spectrum and kinetics of neuroimmune afferent pathways.Leptin regulates leukocyte recruitment into the brain following systemic LPS-induced inflammation.Obesity Impacts Fever and Sickness Behavior During Acute Systemic Inflammation.Role of IL-6 in the etiology of hyperexcitable neuropsychiatric conditions: experimental evidence and therapeutic implications.Distribution of microsomal prostaglandin E synthase-1 in the mouse brain.Fever induction by systemic stimulation with macrophage-activating lipopeptide-2 depends upon TLR2 but not CD36.Spatiotemporal nuclear factor interleukin-6 expression in the rat brain during lipopolysaccharide-induced fever is linked to sustained hypothalamic inflammatory target gene induction.MyD88 plays a key role in LPS-induced Stat3 activation in the hypothalamus.Systemic LPS administration induces brain inflammation but not dopaminergic neuronal death in the substantia nigra.Activation of the inflammatory transcription factor nuclear factor interleukin-6 during inflammatory and psychological stress in the brainThe β2-adrenoceptor agonist clenbuterol reduces the neuroinflammatory response, neutrophil infiltration and apoptosis following intra-striatal IL-1β administration to rats.Role of brain transmigrating neutrophils in depression-like behavior during systemic infection.Leptin Receptor Expression in Mouse Intracranial Perivascular Cells.Age Dependent Hypothalamic and Pituitary Responses to Novel Environment Stress or Lipopolysaccharide in Rats.Neural Mechanisms of Inflammation-Induced Fever
P2860
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P2860
Selective contribution of interleukin-6 and leptin to brain inflammatory signals induced by systemic LPS injection in mice.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Selective contribution of inte ...... ystemic LPS injection in mice.
@en
Selective contribution of inte ...... ystemic LPS injection in mice.
@nl
type
label
Selective contribution of inte ...... ystemic LPS injection in mice.
@en
Selective contribution of inte ...... ystemic LPS injection in mice.
@nl
prefLabel
Selective contribution of inte ...... ystemic LPS injection in mice.
@en
Selective contribution of inte ...... ystemic LPS injection in mice.
@nl
P2093
P2860
P356
P1476
Selective contribution of inte ...... ystemic LPS injection in mice.
@en
P2093
Christelle Sachot
Christoph Rummel
Giamal N Luheshi
Stephen Poole
Thomas Hübschle
Wataru Inoue
P2860
P304
P356
10.1002/CNE.21850
P407
P577
2008-11-01T00:00:00Z