Flurothyl seizure susceptibility in rats following prenatal methylazoxymethanol treatment.
about
Genotoxicants target distinct molecular networks in neonatal neuronsDampened hippocampal oscillations and enhanced spindle activity in an asymptomatic model of developmental cortical malformationsCortical malformations and epilepsy: new insights from animal models.Acute and chronic effects of seizures in the developing brain: experimental models.Cellular abnormalities and synaptic plasticity in seizure disorders of the immature nervous system.Cortical malformations and epilepsy.Cortical dysgenesis and epilepsy.Impaired cognition in rats with cortical dysplasia: additional impact of early-life seizures.Initiation of epileptiform activity in a rat model of periventricular nodular heterotopiaNeuronal migration disorders: heterotopic neocortical neurons in CA1 provide a bridge between the hippocampus and the neocortex.Epilepsy: a review of selected clinical syndromes and advances in basic science.Pathophysiological mechanisms of focal cortical dysplasia: a critical review of human tissue studies and animal models.Neonatal seizures: gaps between the laboratory and the clinic.Decreased glutamate transport enhances excitability in a rat model of cortical dysplasiaDcx reexpression reduces subcortical band heterotopia and seizure threshold in an animal model of neuronal migration disorderCombined effects of prenatal inhibition of vasculogenesis and neurogenesis on rat brain development.Animal models of focal cortical dysplasia and tuberous sclerosis complex: recent progress toward clinical applications.Are developmental dysplastic lesions epileptogenic?Causes and consequences of gray matter heterotopia.Review: Roles for astrocytes in epilepsy: insights from malformations of cortical development.Epileptogenesis in the Dysplastic Brain: A Revival of Familiar ThemesIrradiation exacerbates cortical cytopathology in the Eker rat model of tuberous sclerosis complex, but does not induce hyperexcitability.The etiological role of blood-brain barrier dysfunction in seizure disordersLayer I ectopias and increased excitability in murine neocortex.The NMDA receptor complex is altered in an animal model of human cerebral heterotopia.Prolonged NMDA-mediated responses, altered ifenprodil sensitivity, and epileptiform-like events in the malformed hippocampus of methylazoxymethanol exposed rats.CNS aspects of prenatal drug exposure: drugs of abuse, toxins, and corticosteroids.Neocortex in the hippocampus: an anatomical and functional study of CA1 heterotopias after prenatal treatment with methylazoxymethanol in rats.A computational method for the identification of new candidate carcinogenic and non-carcinogenic chemicals.
P2860
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P2860
Flurothyl seizure susceptibility in rats following prenatal methylazoxymethanol treatment.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh-hant
name
Flurothyl seizure susceptibili ...... methylazoxymethanol treatment.
@en
Flurothyl seizure susceptibili ...... methylazoxymethanol treatment.
@nl
type
label
Flurothyl seizure susceptibili ...... methylazoxymethanol treatment.
@en
Flurothyl seizure susceptibili ...... methylazoxymethanol treatment.
@nl
prefLabel
Flurothyl seizure susceptibili ...... methylazoxymethanol treatment.
@en
Flurothyl seizure susceptibili ...... methylazoxymethanol treatment.
@nl
P1433
P1476
Flurothyl seizure susceptibili ...... methylazoxymethanol treatment.
@en
P2093
P A Schwartzkroin
P304
P356
10.1016/0920-1211(95)00094-1
P577
1996-04-01T00:00:00Z