FTY720 ameliorates acute ischemic stroke in mice by reducing thrombo-inflammation but not by direct neuroprotection. - Wikidata - wikimedia - TriplyDB

FTY720 ameliorates acute ischemic stroke in mice by reducing thrombo-inflammation but not by direct neuroprotection.

FTY720 ameliorates acute ischemic stroke in mice by reducing thrombo-inflammation but not by direct neuroprotection.

http://www.wikidata.org/entity/Q49040217

about

Fingolimod for the treatment of neurological diseases-state of play and future perspectives Partially Defective Store Operated Calcium Entry and Hem(ITAM) Signaling in Platelets of Serotonin Transporter Deficient Mice Essential versus accessory aspects of cell death: recommendations of the NCCD 2015.Fingolimod Limits Acute Aβ Neurotoxicity and Promotes Synaptic Versus Extrasynaptic NMDA Receptor Functionality in Hippocampal Neurons Efficacy of the PSD95 inhibitor Tat-NR2B9c in mice requires dose translation between species.Coadministration of FTY720 and rt-PA in an experimental model of large hemispheric stroke-no influence on functional outcome and blood-brain barrier disruption An update on the biology of sphingosine 1-phosphate receptors.Antigen-specific immune reactions to ischemic stroke.Impact of an immune modulator fingolimod on acute ischemic stroke.Case-control study of platelet glycoprotein receptor Ib and IIb/IIIa expression in patients with acute and chronic cerebrovascular disease.Sphingosine-1-phosphate receptor inhibition prevents denervation-induced dendritic atrophy Platelets induce apoptosis via membrane-bound FasL.Critical role of sphingosine-1-phosphate receptor-2 in the disruption of cerebrovascular integrity in experimental stroke Characterization of Peripheral Immune Cell Subsets in Patients with Acute and Chronic Cerebrovascular Disease: A Case-Control Study Fingolimod effects in neuroinflammation: Regulation of astroglial glutamate transporters?Treatment with FTY720 has no beneficial effects on short-term outcome in an experimental model of intracerebral hemorrhage.Immunohistochemical Analysis of Cerebral Thrombi Retrieved by Mechanical Thrombectomy from Patients with Acute Ischemic Stroke.Mechanism of Action and Clinical Potential of Fingolimod for the Treatment of Stroke.Blocking of platelet glycoprotein receptor Ib reduces "thrombo-inflammation" in mice with acute ischemic stroke.The two-pore domain potassium channel KCNK5 deteriorates outcome in ischemic neurodegeneration.Splenic responses in ischemic stroke: new insights into stroke pathology.Phase II anti-inflammatory and immune-modulating drugs for acute ischaemic stroke.Platelets in inflammation and infection.Animal models of ischemic stroke and their application in clinical research Sphingosine-1-Phosphate Signaling in Endothelial Disorders.Clinical pharmacology, efficacy, and safety aspects of sphingosine-1-phosphate receptor modulators.Sphingosine-1-Phosphate (S1P) and S1P Signaling Pathway: Therapeutic Targets in Autoimmunity and Inflammation.Critical Role of the Sphingolipid Pathway in Stroke: a Review of Current Utility and Potential Therapeutic Targets.Identification of Sphingosine 1-Phosphate Receptor Subtype 1 (S1P1) as a Pathogenic Factor in Transient Focal Cerebral Ischemia.FTY720 attenuates excitotoxicity and neuroinflammation.Therapeutic Potential of Intravenous Immunoglobulin in Acute Brain Injury.Anti-Inflammatory Targets for the Treatment of Reperfusion Injury in Stroke.New monocyte locomotion inhibitory factor analogs protect against cerebral ischemia-reperfusion injury in rats Glatiramer acetate does not protect from acute ischemic stroke in mice.FTY720 elevates smooth muscle contraction of aorta and blood pressure in rats via ERK activation.Microvascular thrombosis, fibrinolysis, ischemic injury, and death after cerebral thromboembolism are affected by levels of circulating α2-antiplasmin.Fingolimod confers neuroprotection through activation of Rac1 after experimental germinal matrix hemorrhage in rat pups.Sphingosine-1-Phosphate Receptor-1 Promotes Environment-Mediated and Acquired Chemoresistance.Fingolimod suppresses neuronal autophagy through the mTOR/p70S6K pathway and alleviates ischemic brain damage in mice.Fingolimod Protects Against Ischemic White Matter Damage by Modulating Microglia Toward M2 Polarization via STAT3 Pathway.

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FTY720 ameliorates acute ischemic stroke in mice by reducing thrombo-inflammation but not by direct neuroprotection.

http://www.wikidata.org/entity/Q49040217

description

2013 nî lūn-bûn

@nan

2013年の論文

@ja

2013年学术文章

@wuu

2013年学术文章

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2013年学术文章

@zh-cn

2013年学术文章

@zh-hans

2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

@zh-hant

name

FTY720 ameliorates acute ische ...... not by direct neuroprotection.

@en

FTY720 ameliorates acute ische ...... not by direct neuroprotection.

@nl

type

label

FTY720 ameliorates acute ische ...... not by direct neuroprotection.

@en

FTY720 ameliorates acute ische ...... not by direct neuroprotection.

@nl

prefLabel

FTY720 ameliorates acute ische ...... not by direct neuroprotection.

@en

FTY720 ameliorates acute ische ...... not by direct neuroprotection.

@nl

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STROKEAHA.113.002880

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FTY720 ameliorates acute ische ...... not by direct neuroprotection.

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Alexander M Herrmann

http://www.w3.org/2001/XMLSchema#string

Bernhard Nieswandt

http://www.w3.org/2001/XMLSchema#string

Eva Göb

http://www.w3.org/2001/XMLSchema#string

Guido Stoll

http://www.w3.org/2001/XMLSchema#string

Ina Thielmann

http://www.w3.org/2001/XMLSchema#string

Kerstin Göbel

http://www.w3.org/2001/XMLSchema#string

Marc Brede

http://www.w3.org/2001/XMLSchema#string

Michael K Schuhmann

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Peter Kraft

http://www.w3.org/2001/XMLSchema#string

Sven G Meuth

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3202-3210

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scholarly article

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10.1161/STROKEAHA.113.002880

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11

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44

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Kristina Lorenz

Carsten Deppermann

Christoph Kleinschnitz

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2013-09-12T00:00:00Z

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24029635

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