In Primary Aldosteronism, Mineralocorticoids Influence Exosomal Sodium-Chloride Cotransporter Abundance.
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NaCl cotransporter abundance in urinary vesicles is increased by calcineurin inhibitors and predicts thiazide sensitivity.Urinary Exosomes and Their Cargo: Potential Biomarkers for Mineralocorticoid Arterial Hypertension?Collecting Duct Nitric Oxide Synthase 1ß Activation Maintains Sodium Homeostasis During High Sodium Intake Through Suppression of Aldosterone and Renal Angiotensin II Pathways.Low-protein diet supplemented with ketoacids delays the progression of diabetic nephropathy by inhibiting oxidative stress in the KKAy mice model.Isolation and characterization of urinary extracellular vesicles: implications for biomarker discovery.Hydrochlorothiazide treatment increases the abundance of the NaCl cotransporter in urinary extracellular vesicles of essential hypertensive patients.Pre-eclampsia is associated with altered expression of the renal sodium transporters NKCC2, NCC and ENaC in urinary extracellular vesiclesRole of the alternative splice variant of NCC in blood pressure control
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P2860
In Primary Aldosteronism, Mineralocorticoids Influence Exosomal Sodium-Chloride Cotransporter Abundance.
description
2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
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name
In Primary Aldosteronism, Mine ...... oride Cotransporter Abundance.
@en
In Primary Aldosteronism, Mine ...... oride Cotransporter Abundance.
@nl
type
label
In Primary Aldosteronism, Mine ...... oride Cotransporter Abundance.
@en
In Primary Aldosteronism, Mine ...... oride Cotransporter Abundance.
@nl
prefLabel
In Primary Aldosteronism, Mine ...... oride Cotransporter Abundance.
@en
In Primary Aldosteronism, Mine ...... oride Cotransporter Abundance.
@nl
P2093
P2860
P356
P1476
In Primary Aldosteronism, Mine ...... oride Cotransporter Abundance.
@en
P2093
Martin J Wolley
Michael Stowasser
Richard D Gordon
Shengxin Xu
P2860
P356
10.1681/ASN.2015111221
P577
2016-07-05T00:00:00Z