MiR-26a-5p regulates cardiac fibroblasts collagen expression by targeting ULK1.

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MiR-26a-5p regulates cardiac fibroblasts collagen expression by targeting ULK1.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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MiR-26a-5p regulates cardiac fibroblasts collagen expression by targeting ULK1.

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MiR-26a-5p regulates cardiac fibroblasts collagen expression by targeting ULK1.

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MiR-26a-5p regulates cardiac fibroblasts collagen expression by targeting ULK1.

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MiR-26a-5p regulates cardiac fibroblasts collagen expression by targeting ULK1.

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MiR-26a-5p regulates cardiac fibroblasts collagen expression by targeting ULK1.

@en

MiR-26a-5p regulates cardiac fibroblasts collagen expression by targeting ULK1.

@nl

P1476

MiR-26a-5p regulates cardiac fibroblasts collagen expression by targeting ULK1.

@en

P2093

Chengyu Lv

http://www.w3.org/2001/XMLSchema#string

Liling Zheng

http://www.w3.org/2001/XMLSchema#string

Sihuang Lin

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P2860

siRNA screening of the kinome identifies ULK1 as a multidomain modulator of autophagy

MicroRNA maturation: stepwise processing and subcellular localization.

LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

MicroRNA expression in response to murine myocardial infarction: miR-21 regulates fibroblast metalloprotease-2 via phosphatase and tensin homologue

The impact of microRNAs on protein output

Mammalian microRNAs predominantly act to decrease target mRNA levels

The C. elegans heterochronic gene lin-4 encodes small RNAs with antisense complementarity to lin-14

Autophagy: renovation of cells and tissues

Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis

STAT3 down regulates LC3 to inhibit autophagy and pancreatic cancer cell growth.

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2104

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P31

scholarly article

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10.1038/S41598-018-20561-4

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English

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P478

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P577

2018-02-01T00:00:00Z

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29391491

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5794903

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