IDH1R132H Promotes Malignant Transformation of Benign Prostatic Epithelium by Dysregulating MicroRNAs: Involvement of IGF1R-AKT/STAT3 Signaling Pathway.

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IDH1R132H Promotes Malignant Transformation of Benign Prostatic Epithelium by Dysregulating MicroRNAs: Involvement of IGF1R-AKT/STAT3 Signaling Pathway.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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name

IDH1R132H Promotes Malignant T ...... R-AKT/STAT3 Signaling Pathway.

@en

IDH1R132H Promotes Malignant T ...... R-AKT/STAT3 Signaling Pathway.

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type

Item

label

IDH1R132H Promotes Malignant T ...... R-AKT/STAT3 Signaling Pathway.

@en

IDH1R132H Promotes Malignant T ...... R-AKT/STAT3 Signaling Pathway.

@nl

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IDH1R132H Promotes Malignant T ...... R-AKT/STAT3 Signaling Pathway.

@en

IDH1R132H Promotes Malignant T ...... R-AKT/STAT3 Signaling Pathway.

@nl

P1476

IDH1R132H Promotes Malignant T ...... R-AKT/STAT3 Signaling Pathway.

@en

P2093

Bo Han

http://www.w3.org/2001/XMLSchema#string

Hui Liu

http://www.w3.org/2001/XMLSchema#string

Jing Hu

http://www.w3.org/2001/XMLSchema#string

Lili Zhang

http://www.w3.org/2001/XMLSchema#string

Lin Wang

http://www.w3.org/2001/XMLSchema#string

Long Liu

http://www.w3.org/2001/XMLSchema#string

Mei Qi

http://www.w3.org/2001/XMLSchema#string

Meng Jiao

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Tingting Feng

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Wei Gao

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P2860

Real-time expression profiling of microRNA precursors in human cancer cell lines.

Tissue biomarkers for prognosis of prostate cancer: a systematic review and meta-analysis

An inhibitor of mutant IDH1 delays growth and promotes differentiation of glioma cells.

Expression of the type 1 insulin-like growth factor receptor is up-regulated in primary prostate cancer and commonly persists in metastatic disease

Insulin-like growth factors and their binding proteins: biological actions

IDH mutation impairs histone demethylation and results in a block to cell differentiation

IDH1 mutation is sufficient to establish the glioma hypermethylator phenotype

Autocrine IGF-1/IGF-1R signaling is responsible for constitutive PI3K/Akt activation in acute myeloid leukemia: therapeutic value of neutralizing anti-IGF-1R antibody

IDH1 and IDH2 gene mutations identify novel molecular subsets within de novo cytogenetically normal acute myeloid leukemia: a Cancer and Leukemia Group B study.

Molecular genetics of prostate cancer: new prospects for old challenges

P304

207-217

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scholarly article

P356

10.1016/J.NEO.2017.12.001

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P478

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2018-01-11T00:00:00Z

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P698

29331887

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P921

microRNA

P932

5767912

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