Overexpression of CREPT confers colorectal cancer sensitivity to fluorouracil.

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Overexpression of CREPT confers colorectal cancer sensitivity to fluorouracil.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Overexpression of CREPT confers colorectal cancer sensitivity to fluorouracil.

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Overexpression of CREPT confers colorectal cancer sensitivity to fluorouracil.

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Overexpression of CREPT confers colorectal cancer sensitivity to fluorouracil.

@en

Overexpression of CREPT confers colorectal cancer sensitivity to fluorouracil.

@nl

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Overexpression of CREPT confers colorectal cancer sensitivity to fluorouracil.

@en

Overexpression of CREPT confers colorectal cancer sensitivity to fluorouracil.

@nl

P1476

Overexpression of CREPT confers colorectal cancer sensitivity to fluorouracil.

@en

P2093

Bao-Qing Jia

http://www.w3.org/2001/XMLSchema#string

Bing-Tao Zhu

http://www.w3.org/2001/XMLSchema#string

Hong-Yi Liu

http://www.w3.org/2001/XMLSchema#string

Jun Li

http://www.w3.org/2001/XMLSchema#string

Li-Dan Ding

http://www.w3.org/2001/XMLSchema#string

Liu Yang

http://www.w3.org/2001/XMLSchema#string

Si-Han Liu

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Xu-Ning Wang

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Yan-Shen Kuang

http://www.w3.org/2001/XMLSchema#string

Yi Wang

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P2860

CREPT accelerates tumorigenesis by regulating the transcription of cell-cycle-related genes

5-fluorouracil: mechanisms of action and clinical strategies

miR-1290 Is a Biomarker in DNA-Mismatch-Repair-Deficient Colon Cancer and Promotes Resistance to 5-Fluorouracil by Directly Targeting hMSH2.

CREPT/RPRD1B, a recently identified novel protein highly expressed in tumors, enhances the β-catenin·TCF4 transcriptional activity in response to Wnt signaling.

CREPT expression correlates with poor prognosis in patients with retroperitoneal leiomyosarcoma.

Colorectal cancer: current imaging methods and future perspectives for the diagnosis, staging and therapeutic response evaluation.

A review of the evolution of systemic chemotherapy in the management of colorectal cancer.

MiR-1188 at the imprinted Dlk1-Dio3 domain acts as a tumor suppressor in hepatoma cells.

A peptide of SPARC interferes with the interaction between caspase8 and Bcl2 to resensitize chemoresistant tumors and enhance their regression in vivo

Knocking-down of CREPT prohibits the progression of oral squamous cell carcinoma and suppresses cyclin D1 and c-Myc expression.

P304

475-483

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P31

scholarly article

P356

10.3748/WJG.V24.I4.475

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2018-01-01T00:00:00Z

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P698

29398868

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P921

colorectal cancer

overexpression

P932

5787782

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