Multiple signaling kinases target Mrc1 to prevent genomic instability triggered by transcription-replication conflicts.

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Multiple signaling kinases target Mrc1 to prevent genomic instability triggered by transcription-replication conflicts.

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2018 nî lūn-bûn

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2018年学术文章

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Multiple signaling kinases tar ...... ription-replication conflicts.

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Multiple signaling kinases tar ...... ription-replication conflicts.

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Multiple signaling kinases tar ...... ription-replication conflicts.

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Multiple signaling kinases tar ...... ription-replication conflicts.

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Multiple signaling kinases tar ...... ription-replication conflicts.

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Multiple signaling kinases tar ...... ription-replication conflicts.

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Multiple signaling kinases tar ...... cription-replication conflicts

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Alba Duch

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Berta Canal

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Eulàlia de Nadal

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Gerhard Seisenbacher

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María García-Rubio

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Sonia I Barroso

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P2860

Global analysis of protein expression in yeast

Genomic expression programs in the response of yeast cells to environmental changes

APC(Cdc20) promotes exit from mitosis by destroying the anaphase inhibitor Pds1 and cyclin Clb5.

Regulation of Snf1 kinase. Activation requires phosphorylation of threonine 210 by an upstream kinase as well as a distinct step mediated by the Snf4 subunit.

Control of Cdc28 CDK1 by a stress-induced lncRNA

Remodeling of yeast genome expression in response to environmental changes.

Hog1 bypasses stress-mediated down-regulation of transcription by RNA polymerase II redistribution and chromatin remodeling

Mrc1 transduces signals of DNA replication stress to activate Rad53.

Yeast SNF1 is functionally related to mammalian AMP-activated protein kinase and regulates acetyl-CoA carboxylase in vivo.

Testing cyclin specificity in the exit from mitosis.

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s41467-017-02756-x

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scholarly article

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10.1038/S41467-017-02756-X

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English

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Andrés Aguilera

Francesc Posas

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2018-01-25T00:00:00Z

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1100522950

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29371596

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5785523

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