Cardiomyocyte-specific deletion of GSK-3β leads to cardiac dysfunction in a diet induced obesity model.

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Cardiomyocyte-specific deletion of GSK-3β leads to cardiac dysfunction in a diet induced obesity model.

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2018 nî lūn-bûn

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Cardiomyocyte-specific deletio ...... a diet induced obesity model.

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Cardiomyocyte-specific deletio ...... a diet induced obesity model.

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Cardiomyocyte-specific deletio ...... a diet induced obesity model.

@en

Cardiomyocyte-specific deletio ...... a diet induced obesity model.

@nl

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Cardiomyocyte-specific deletio ...... a diet induced obesity model.

@en

Cardiomyocyte-specific deletio ...... a diet induced obesity model.

@nl

P1476

Cardiomyocyte-specific deletio ...... n a diet induced obesity model

@en

P2093

Jennifer Y Sui

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Manisha Gupte

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Samvruta Tumuluru

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Thomas Force

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P2860

The GSK-3 family as therapeutic target for myocardial diseases

Global prevalence of diabetes: estimates for the year 2000 and projections for 2030

Stabilised beta-catenin in postnatal ventricular myocardium leads to dilated cardiomyopathy and premature death

Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale.

Beta-catenin downregulation is required for adaptive cardiac remodeling

Selumetinib, an Oral Anti-Neoplastic Drug, May Attenuate Cardiac Hypertrophy via Targeting the ERK Pathway

GSK-3alpha directly regulates beta-adrenergic signaling and the response of the heart to hemodynamic stress in mice

Chronic high fat diet induces cardiac hypertrophy and fibrosis in mice.

Glycogen synthase kinase-3beta regulates post-myocardial infarction remodeling and stress-induced cardiomyocyte proliferation in vivo

Cardiomyocyte-specific deletion of Gsk3α mitigates post-myocardial infarction remodeling, contractile dysfunction, and heart failure.

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145-152

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scholarly article

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10.1016/J.IJCARD.2018.01.013

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English

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259

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P577

2018-02-03T00:00:00Z

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29398139

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Adipositas

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5869114

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