Hypercapnic Acidosis Regulates Mer Tyrosine Kinase Receptor Shedding and Activity.

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Hypercapnic Acidosis Regulates Mer Tyrosine Kinase Receptor Shedding and Activity.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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2018年學術文章

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Hypercapnic Acidosis Regulates Mer Tyrosine Kinase Receptor Shedding and Activity.

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Hypercapnic Acidosis Regulates Mer Tyrosine Kinase Receptor Shedding and Activity.

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Hypercapnic Acidosis Regulates Mer Tyrosine Kinase Receptor Shedding and Activity.

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Hypercapnic Acidosis Regulates Mer Tyrosine Kinase Receptor Shedding and Activity.

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Hypercapnic Acidosis Regulates Mer Tyrosine Kinase Receptor Shedding and Activity.

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Hypercapnic Acidosis Regulates Mer Tyrosine Kinase Receptor Shedding and Activity.

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P1476

Hypercapnic Acidosis Regulates Mer Tyrosine Kinase Receptor Shedding and Activity.

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P2093

Brian P Kavanagh

http://www.w3.org/2001/XMLSchema#string

Claire Masterson

http://www.w3.org/2001/XMLSchema#string

Doreen Engelberts

http://www.w3.org/2001/XMLSchema#string

Gail Otulakowski

http://www.w3.org/2001/XMLSchema#string

Martin Post

http://www.w3.org/2001/XMLSchema#string

P2860

Acidification prevents endothelial cell apoptosis by Axl activation

Shedding of the Mer tyrosine kinase receptor is mediated by ADAM17 protein through a pathway involving reactive oxygen species, protein kinase Cδ, and p38 mitogen-activated protein kinase (MAPK).

Can 'permissive' hypercapnia modulate the severity of sepsis-induced ALI/ARDS?

A soluble form of the Mer receptor tyrosine kinase inhibits macrophage clearance of apoptotic cells and platelet aggregation.

TAM receptor signaling in immune homeostasis.

MerTK cleavage limits proresolving mediator biosynthesis and exacerbates tissue inflammation.

Growth arrest-specific protein 6 attenuates neutrophil migration and acute lung injury in sepsis

Has mortality from acute respiratory distress syndrome decreased over time?: A systematic review.

Activation of NLRP3 inflammasome in alveolar macrophages contributes to mechanical stretch-induced lung inflammation and injury.

Plasma concentrations of Gas6 (growth arrest specific protein 6) and its soluble tyrosine kinase receptor sAxl in sepsis and systemic inflammatory response syndromes.

P304

132-134

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P31

scholarly article

P356

10.1165/RCMB.2017-0316LE

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2018-01-01T00:00:00Z

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P698

29286859

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P921

hypercapnia