Polymorphonuclear Leukocytes in Pulmonary Hypertension and Fibrosis: Not Always What They Appear to Be.

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Polymorphonuclear Leukocytes in Pulmonary Hypertension and Fibrosis: Not Always What They Appear to Be.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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Polymorphonuclear Leukocytes i ...... Always What They Appear to Be.

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Polymorphonuclear Leukocytes i ...... Always What They Appear to Be.

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label

Polymorphonuclear Leukocytes i ...... Always What They Appear to Be.

@en

Polymorphonuclear Leukocytes i ...... Always What They Appear to Be.

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Polymorphonuclear Leukocytes i ...... Always What They Appear to Be.

@en

Polymorphonuclear Leukocytes i ...... Always What They Appear to Be.

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P1476

Polymorphonuclear Leukocytes i ...... Always What They Appear to Be.

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P2093

Gabriele Grunig

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Nedim Durmus

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P2860

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Interleukin 13- and interleukin 17A-induced pulmonary hypertension phenotype due to inhalation of antigen and fine particles from air pollution

Circulating myeloid-derived suppressor cells are increased and activated in pulmonary hypertension.

The Causal Role of IL-4 and IL-13 in Schistosoma mansoni Pulmonary Hypertension.

Idiopathic pulmonary fibrosis and pulmonary hypertension: connecting the dots.

HIF2α-arginase axis is essential for the development of pulmonary hypertension.

Diesel exhaust particle induction of IL-17A contributes to severe asthma.

P304

135-137

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scholarly article

editorial

P356

10.1165/RCMB.2017-0336ED

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P433

http://www.w3.org/2001/XMLSchema#string

P478

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2018-02-01T00:00:00Z

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29388835

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P932

5806001

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