Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.

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LIGHT/TNFSF14 as a New Biomarker of Bone Disease in Multiple Myeloma Patients Experiencing Therapeutic Regimens

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Q58572512-14970E3B-7498-463D-A8A6-8E41B716D985

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Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.

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http://www.wikidata.org/entity/Q49602216

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2017年学术文章

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Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.

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Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.

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type

Item

label

Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.

@en

Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.

@nl

prefLabel

Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.

@en

Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.

@nl

P1476

Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.

@en

P2093

Adriana Di Benedetto

http://www.w3.org/2001/XMLSchema#string

Angela Oranger

http://www.w3.org/2001/XMLSchema#string

Carl F Ware

http://www.w3.org/2001/XMLSchema#string

Ernestina Schipani

http://www.w3.org/2001/XMLSchema#string

Giovanni Passeri

http://www.w3.org/2001/XMLSchema#string

Giuseppina Storlino

http://www.w3.org/2001/XMLSchema#string

Graziana Colaianni

http://www.w3.org/2001/XMLSchema#string

Isabella Gigante

http://www.w3.org/2001/XMLSchema#string

Janne E Reseland

http://www.w3.org/2001/XMLSchema#string

Koji Tamada

http://www.w3.org/2001/XMLSchema#string

P2860

Herpesvirus entry mediator ligand (HVEM-L), a novel ligand for HVEM/TR2, stimulates proliferation of T cells and inhibits HT29 cell growth

LIGHT, a new member of the TNF superfamily, and lymphotoxin alpha are ligands for herpesvirus entry mediator

T lymphocytes amplify the anabolic activity of parathyroid hormone through Wnt10b signaling

Herpesvirus entry mediator, a member of the tumor necrosis factor receptor (TNFR) family, interacts with members of the TNFR-associated factor family and activates the transcription factors NF-kappaB and AP-1

osteoprotegerin-deficient mice develop early onset osteoporosis and arterial calcification

The Lymphotoxin Network: orchestrating a type I interferon response to optimize adaptive immunity

A newly identified member of the tumor necrosis factor receptor superfamily with a wide tissue distribution and involvement in lymphocyte activation

Osteoimmunology: shared mechanisms and crosstalk between the immune and bone systems

OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis

RAG-1-deficient mice have no mature B and T lymphocytes

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704-719

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scholarly article

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10.1002/JBMR.3345

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

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Giacomina Brunetti

Giorgio Mori

Giuseppe Ingravallo

P577

2017-11-27T00:00:00Z

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P698

29178458

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Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.

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P2860

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Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.

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P1433

P1476

P2093

P2860

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P31

P356

P433

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P50

P577

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698