about
Lipopolysaccharide-induced NF-κB nuclear translocation is primarily dependent on MyD88, but TNFα expression requires TRIF and MyD88Actin polymerization as a key innate immune effector mechanism to control Salmonella infectionThe frequency and duration of Salmonella-macrophage adhesion events determines infection efficiencyInflammasome activation causes dual recruitment of NLRC4 and NLRP3 to the same macromolecular complex.CARD9 negatively regulates NLRP3-induced IL-1β production on Salmonella infection of macrophages.Interaction of staphylococci with bone.An aroD Ochre Mutation Results in a Staphylococcus aureus Small Colony Variant That Can Undergo Phenotypic Switching via Two Alternative Mechanisms.Staphylococcus aureus small colony variants are susceptible to light activated antimicrobial agents.Multiple redundant stress resistance mechanisms are induced in Salmonella enterica serovar Typhimurium in response to alteration of the intracellular environment via TLR4 signalling.Metabolite and transcriptome analysis of Campylobacter jejuni in vitro growth reveals a stationary-phase physiological switch.Influence of type-I fimbriae and fluid shear stress on bacterial behavior and multicellular architecture of early Escherichia coli biofilms at single-cell resolution.Bicarbonate induces high-level resistance to the human antimicrobial peptide LL-37 in Staphylococcus aureus small colony variants.The killer protein Gasdermin D.Comparative genome analysis of Campylobacter jejuni using whole genome DNA microarrays.Toll-like receptor 3 activation impairs excitability and synaptic activity via TRIF signalling in immature rat and human neurons.
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
John A Wright
@ast
John A Wright
@en
John A Wright
@es
John A Wright
@nl
John A Wright
@sl
type
label
John A Wright
@ast
John A Wright
@en
John A Wright
@es
John A Wright
@nl
John A Wright
@sl
prefLabel
John A Wright
@ast
John A Wright
@en
John A Wright
@es
John A Wright
@nl
John A Wright
@sl
P106
P31
P496
0000-0002-9758-9944