MiR-34c induces apoptosis and inhibits the viability of M4e cells by targeting BCL2.

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MiR-34c induces apoptosis and inhibits the viability of M4e cells by targeting BCL2.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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MiR-34c induces apoptosis and inhibits the viability of M4e cells by targeting BCL2.

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MiR-34c induces apoptosis and inhibits the viability of M4e cells by targeting BCL2.

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MiR-34c induces apoptosis and inhibits the viability of M4e cells by targeting BCL2.

@en

MiR-34c induces apoptosis and inhibits the viability of M4e cells by targeting BCL2.

@nl

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MiR-34c induces apoptosis and inhibits the viability of M4e cells by targeting BCL2.

@en

MiR-34c induces apoptosis and inhibits the viability of M4e cells by targeting BCL2.

@nl

P1476

MiR-34c induces apoptosis and inhibits the viability of M4e cells by targeting BCL2.

@en

P2093

Hongxia Zhang

http://www.w3.org/2001/XMLSchema#string

Rui Li

http://www.w3.org/2001/XMLSchema#string

Xiling Zheng

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P2860

miR-15 and miR-16 induce apoptosis by targeting BCL2

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MicroRNA-27a promotes proliferation and suppresses apoptosis by targeting PLK2 in laryngeal carcinoma

KITLG is a novel target of miR-34c that is associated with the inhibition of growth and invasion in colorectal cancer cells.

MicroRNA-196a is a putative diagnostic biomarker and therapeutic target for laryngeal cancer.

MicroRNA-16 inhibits glioma cell growth and invasion through suppression of BCL2 and the nuclear factor-κB1/MMP9 signaling pathway.

Gene and protein expression of glucose transporter 1 and glucose transporter 3 in human laryngeal cancer-the relationship with regulatory hypoxia-inducible factor-1α expression, tumor invasiveness, and patient prognosis.

MicroRNA dysregulation in cancer: diagnostics, monitoring and therapeutics. A comprehensive review.

microRNA and gene networks in human laryngeal cancer.

Galangin suppresses human laryngeal carcinoma via modulation of caspase-3 and AKT signaling pathways.

P304

3357-3361

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P31

scholarly article

P356

10.3892/OL.2017.7640

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P433

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P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-12-19T00:00:00Z

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P698

29435079

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P932

5778784

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