Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase.
about
NO-mediated cytoprotection: instant adaptation to oxidative stress in bacteria.Effects of Anaplasma phagocytophila on NADPH oxidase components in human neutrophils and HL-60 cells.Infection-related hemolysis and susceptibility to Gram-negative bacterial co-infectionMucosal Inflammatory Response to Salmonella typhimurium InfectionInteractions of Salmonella enterica with dendritic cellsWhat really happens in the neutrophil phagosome?The Dynamic Interactions between Salmonella and the Microbiota, within the Challenging Niche of the Gastrointestinal TractPeroxynitrite, a potent macrophage-derived oxidizing cytotoxin to combat invading pathogensNADPH oxidase deficient mice develop colitis and bacteremia upon infection with normally avirulent, TTSS-1- and TTSS-2-deficient Salmonella TyphimuriumEthanolamine Signaling Promotes Salmonella Niche Recognition and Adaptation during Infection.Transcriptional priming of Salmonella Pathogenicity Island-2 precedes cellular invasionAttenuated Salmonella Typhimurium lacking the pathogenicity island-2 type 3 secretion system grow to high bacterial numbers inside phagocytes in miceCellular aspects of immunity to intracellular Salmonella entericaHow does the oxidative burst of macrophages kill bacteria? Still an open questionThe bacterial cytoskeleton modulates motility, type 3 secretion, and colonization in SalmonellaIdentification of Mycobacterium tuberculosis counterimmune (cim) mutants in immunodeficient mice by differential screeningThe inflammation-associated Salmonella SopA is a HECT-like E3 ubiquitin ligaseSalmonella pathogenicity island 2-encoded proteins SseC and SseD are essential for virulence and are substrates of the type III secretion systemSopD2 is a novel type III secreted effector of Salmonella typhimurium that targets late endocytic compartments upon delivery into host cellsAutophagy proteins control goblet cell function by potentiating reactive oxygen species productionEffect of immune serum and role of individual Fcgamma receptors on the intracellular distribution and survival of Salmonella enterica serovar Typhimurium in murine macrophagesVirulence comparisons of Aspergillus nidulans mutants are confounded by the inflammatory response of p47phox-/- mice.Salmonella enterica serovar Typhimurium periplasmic superoxide dismutase SodCI is a member of the PhoPQ regulon and is induced in macrophagesThe phosphoinositide-binding protein p40phox activates the NADPH oxidase during FcgammaIIA receptor-induced phagocytosisRegulation of Rac1 and Reactive Oxygen Species Production in Response to Infection of Gastrointestinal EpitheliaFlagella facilitate escape of Salmonella from oncotic macrophages.Dynamics of intracellular bacterial replication at the single cell level.Dynamics of Salmonella infection of macrophages at the single cell level.Direct measurement of oxidative and nitrosative stress dynamics in Salmonella inside macrophagesExamination of Salmonella gene expression in an infected mammalian host using the green fluorescent protein and two-colour flow cytometry.Molecular insights into farm animal and zoonotic Salmonella infections.Methionine sulfoxide reductase A (MsrA) deficiency affects the survival of Mycobacterium smegmatis within macrophages.sciS, an icmF homolog in Salmonella enterica serovar Typhimurium, limits intracellular replication and decreases virulenceThe Francisella pathogenicity island protein IglA localizes to the bacterial cytoplasm and is needed for intracellular growthEvasion of Legionella pneumophila from the bactericidal system by reactive oxygen species (ROS) in macrophages.RpoE fine tunes expression of a subset of SsrB-regulated virulence factors in Salmonella enterica serovar Typhimurium.lac repressor is an antivirulence factor of Salmonella enterica: its role in the evolution of virulence in Salmonella.Division of the Salmonella-containing vacuole and depletion of acidic lysosomes in Salmonella-infected host cells are novel strategies of Salmonella enterica to avoid lysosomesCurcumin increases the pathogenicity of Salmonella enterica serovar Typhimurium in murine model.Microarray analysis of response of Salmonella during infection of HLA-B27- transfected human macrophage-like U937 cells.
P2860
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P2860
Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年学术文章
@wuu
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
@yue
2000年學術文章
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2000年學術文章
@zh-hant
name
Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase.
@en
Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase.
@nl
type
label
Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase.
@en
Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase.
@nl
prefLabel
Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase.
@en
Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase.
@nl
P2093
P1433
P1476
Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase.
@en
P2093
Dinauer MC
Jones-Carson J
Mastroeni P
Vazquez-Torres A
P304
P356
10.1126/SCIENCE.287.5458.1655
P407
P50
P577
2000-03-01T00:00:00Z