Requirement of CDC42 for Salmonella-induced cytoskeletal and nuclear responses.
about
Interaction of the Salmonella Typhimurium effector protein SopB with host cell Cdc42 is involved in intracellular replicationStructural basis for the reversible activation of a Rho protein by the bacterial toxin SopE.Actin-based motility of intracellular microbial pathogensType III protein secretion systems in bacterial pathogens of animals and plantsSGEF, a RhoG guanine nucleotide exchange factor that stimulates macropinocytosis.Common themes in microbial pathogenicity revisitedInvestigating the function of Rho family GTPases during Salmonella/host cell interactionsDifferential activation and function of Rho GTPases during Salmonella-host cell interactionsFlagellin acting via TLR5 is the major activator of key signaling pathways leading to NF-kappa B and proinflammatory gene program activation in intestinal epithelial cellsA Family of Salmonella Type III Secretion Effector Proteins Selectively Targets the NF-κB Signaling Pathway to Preserve Host HomeostasisSalmonella Typhimurium type III secretion effectors stimulate innate immune responses in cultured epithelial cellsCdc42: An essential Rho-type GTPase controlling eukaryotic cell polarity.Macropinocytosis Exploitation by Cancers and Cancer TherapeuticsInvolvement of focal adhesion kinase in invasin-mediated uptakePhenotypic pattern-based assay for dynamically monitoring host cellular responses to Salmonella infectionsFine tuning inflammation at the front door: macrophage complement receptor 3-mediates phagocytosis and immune suppression for Francisella tularensisRole of SPI-1 secreted effectors in acute bovine response to Salmonella enterica Serovar Typhimurium: a systems biology analysis approachWAVE2 signaling mediates invasion of polarized epithelial cells by Salmonella typhimurium.Requirement of p21-activated kinase (PAK) for Salmonella typhimurium-induced nuclear responses.Coordinate regulation of Salmonella enterica serovar Typhimurium invasion of epithelial cells by the Arp2/3 complex and Rho GTPases.Invasion of host cells by Salmonella typhimurium requires focal adhesion kinase and p130CasHelicobacter pylori CagA induces AGS cell elongation through a cell retraction defect that is independent of Cdc42, Rac1, and Arp2/3.Salmonella enterica serovar typhimurium pathogenicity island 1-encoded type III secretion system translocases mediate intimate attachment to nonphagocytic cellsAspect ratio determines the quantity of mesoporous silica nanoparticle uptake by a small GTPase-dependent macropinocytosis mechanism.Salmonella enterica Serovar Typhi conceals the invasion-associated type three secretion system from the innate immune system by gene regulation.A Salmonella inositol polyphosphatase acts in conjunction with other bacterial effectors to promote host cell actin cytoskeleton rearrangements and bacterial internalization.Deciphering interplay between Salmonella invasion effectors.SopE and SopE2 from Salmonella typhimurium activate different sets of RhoGTPases of the host cell.Signaling pathways in phagocytosis.Cell-contact-stimulated formation of filamentous appendages by Salmonella typhimurium does not depend on the type III secretion system encoded by Salmonella pathogenicity island 1.Inhibition of Salmonella typhimurium invasion by host cell expression of secreted bacterial invasion proteinsInvasion of epithelial cells by locus of enterocyte effacement-negative enterohemorrhagic Escherichia coliLive attenuated Salmonella: a paradigm of mucosal vaccines.Antagonistic activity of Lactobacillus acidophilus LB against intracellular Salmonella enterica serovar Typhimurium infecting human enterocyte-like Caco-2/TC-7 cellsDifferential regulation of Salmonella typhimurium type III secreted proteins by pathogenicity island 1 (SPI-1)-encoded transcriptional activators InvF and hilA.The arginine finger domain of ExoT contributes to actin cytoskeleton disruption and inhibition of internalization of Pseudomonas aeruginosa by epithelial cells and macrophages.Opsonization modulates Rac-1 activation during cell entry by Leishmania amazonensis.Pathogen-induced apoptosis of macrophages: a common end for different pathogenic strategies.Efficient Salmonella entry requires activity cycles of host ADF and cofilin.The phosphatidylserine receptor: a crucial molecular switch?
P2860
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P2860
Requirement of CDC42 for Salmonella-induced cytoskeletal and nuclear responses.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh-hant
name
Requirement of CDC42 for Salmonella-induced cytoskeletal and nuclear responses.
@en
Requirement of CDC42 for Salmonella-induced cytoskeletal and nuclear responses.
@nl
type
label
Requirement of CDC42 for Salmonella-induced cytoskeletal and nuclear responses.
@en
Requirement of CDC42 for Salmonella-induced cytoskeletal and nuclear responses.
@nl
prefLabel
Requirement of CDC42 for Salmonella-induced cytoskeletal and nuclear responses.
@en
Requirement of CDC42 for Salmonella-induced cytoskeletal and nuclear responses.
@nl
P2093
P2860
P1433
P1476
Requirement of CDC42 for Salmonella-induced cytoskeletal and nuclear responses.
@en
P2093
P2860
P304
P356
10.1126/SCIENCE.274.5295.2115
P407
P577
1996-12-01T00:00:00Z