Underlying mechanism and specific prevention of hemolysis-induced platelet activation.

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Decreased platelet inhibition by P2Y12 receptor blockers in anaemia.Elevated free hemoglobin and decreased haptoglobin levels are associated with adverse clinical outcomes, unfavorable physiologic measures, and altered inflammatory markers in pediatric cardiac surgery patients.

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Underlying mechanism and specific prevention of hemolysis-induced platelet activation.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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name

Underlying mechanism and specific prevention of hemolysis-induced platelet activation.

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Underlying mechanism and specific prevention of hemolysis-induced platelet activation.

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Underlying mechanism and specific prevention of hemolysis-induced platelet activation.

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Underlying mechanism and specific prevention of hemolysis-induced platelet activation.

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Underlying mechanism and specific prevention of hemolysis-induced platelet activation.

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Underlying mechanism and specific prevention of hemolysis-induced platelet activation.

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P1476

Underlying mechanism and specific prevention of hemolysis-induced platelet activation.

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P2093

Beate Eichelberger

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Günter Weigel

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Simon Panzer

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Simone Fedrizzi

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P2860

Role of CD39 (NTPDase-1) in thromboregulation, cerebroprotection, and cardioprotection

Ex vivo evaluation of erythrocytosis-enhanced platelet thrombus formation using the cone and plate(let) analyzer: effect of platelet antagonists

Adenosine diphosphate in red cells as a factor in the adhesiveness of human blood platelets.

The clinical sequelae of intravascular hemolysis and extracellular plasma hemoglobin: a novel mechanism of human disease.

The hypercoagulable state in thalassemia.

Auto-immune haemolytic anaemia--a high-risk disorder for thromboembolism?

Free hemoglobin increases von Willebrand factor-mediated platelet adhesion in vitro: implications for circulatory devices.

Hemoglobin interaction with GP1bα induces platelet activation and apoptosis: a novel mechanism associated with intravascular hemolysis.

A double-blind, randomized, multicenter phase 2 study of prasugrel versus placebo in adult patients with sickle cell disease.

Beta-thalassaemia and sickle cell anaemia as paradigms of hypercoagulability.

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555-559

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scholarly article

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10.1080/09537104.2016.1240769

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P478

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Thomas Gremmel

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2016-11-16T00:00:00Z

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27848264

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Underlying mechanism and specific prevention of hemolysis-induced platelet activation.

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P2860

P2860

Underlying mechanism and specific prevention of hemolysis-induced platelet activation.

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P1476

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P2860

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P698