KIND1 Loss Sensitizes Keratinocytes to UV-Induced Inflammatory Response and DNA Damage.

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KIND1 Loss Sensitizes Keratinocytes to UV-Induced Inflammatory Response and DNA Damage.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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KIND1 Loss Sensitizes Keratinocytes to UV-Induced Inflammatory Response and DNA Damage.

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KIND1 Loss Sensitizes Keratinocytes to UV-Induced Inflammatory Response and DNA Damage.

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KIND1 Loss Sensitizes Keratinocytes to UV-Induced Inflammatory Response and DNA Damage.

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KIND1 Loss Sensitizes Keratinocytes to UV-Induced Inflammatory Response and DNA Damage.

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KIND1 Loss Sensitizes Keratinocytes to UV-Induced Inflammatory Response and DNA Damage.

@en

KIND1 Loss Sensitizes Keratinocytes to UV-Induced Inflammatory Response and DNA Damage.

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P1476

KIND1 Loss Sensitizes Keratinocytes to UV-Induced Inflammatory Response and DNA Damage.

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P2093

Detlev Erdmann

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Jennifer Y Zhang

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Joseph Wu

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Long Zhang

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Russell Hall

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Simone Degan

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Suju Luo

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Wen-Hui Wang

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Xiaoling Zhang

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P2860

Kindlin-1 is a phosphoprotein involved in regulation of polarity, proliferation, and motility of epidermal keratinocytes

A comprehensive catalogue of somatic mutations from a human cancer genome

Serpin squamous cell carcinoma antigen inhibits UV-induced apoptosis via suppression of c-JUN NH2-terminal kinase

Molecular mechanisms of epithelial-mesenchymal transition

Kindlin-1 regulates integrin dynamics and adhesion turnover

TNF-R1 signaling: a beautiful pathway

SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase

Kindlin-1 controls Wnt and TGF-β availability to regulate cutaneous stem cell proliferation

NF-kappaB: linking inflammation and immunity to cancer development and progression

Identification of an oncoprotein- and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain

P304

475-483

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P31

scholarly article

P356

10.1016/J.JID.2016.09.023

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P407

English

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P478

137

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P577

2016-10-07T00:00:00Z

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P698

27725201

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P932

5258776

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