Aryl Hydrocarbon Receptor Activation Modulates Intestinal Epithelial Barrier Function by Maintaining Tight Junction Integrity.

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Aryl Hydrocarbon Receptor Activation Modulates Intestinal Epithelial Barrier Function by Maintaining Tight Junction Integrity.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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Aryl Hydrocarbon Receptor Acti ...... ning Tight Junction Integrity.

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Aryl Hydrocarbon Receptor Acti ...... ning Tight Junction Integrity.

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Aryl Hydrocarbon Receptor Acti ...... ning Tight Junction Integrity.

@en

Aryl Hydrocarbon Receptor Acti ...... ning Tight Junction Integrity.

@nl

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Aryl Hydrocarbon Receptor Acti ...... ning Tight Junction Integrity.

@en

Aryl Hydrocarbon Receptor Acti ...... ning Tight Junction Integrity.

@nl

P1476

Aryl Hydrocarbon Receptor Acti ...... ning Tight Junction Integrity.

@en

P2093

Guoqing Chen

http://www.w3.org/2001/XMLSchema#string

Hua Yang

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Kun Yu

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Liangzi Li

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Min Yu

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Pengyuan Xu

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Qimeng Wang

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Weidong Xiao

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Xiangsheng Li

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Yuanhang Ma

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P2860

Interferon-gamma and tumor necrosis factor-alpha synergize to induce intestinal epithelial barrier dysfunction by up-regulating myosin light chain kinase expression.

Aryl hydrocarbon receptor-induced signals up-regulate IL-22 production and inhibit inflammation in the gastrointestinal tract

Exogenous stimuli maintain intraepithelial lymphocytes via aryl hydrocarbon receptor activation

Tumor necrosis factor-induced long myosin light chain kinase transcription is regulated by differentiation-dependent signaling events. Characterization of the human long myosin light chain kinase promoter

Anti-androgen flutamide suppresses hepatocellular carcinoma cell proliferation via the aryl hydrocarbon receptor mediated induction of transforming growth factor-β1

The aryl hydrocarbon receptor complex and the control of gene expression

Exactly the same but different: promiscuity and diversity in the molecular mechanisms of action of the aryl hydrocarbon (dioxin) receptor

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

Amelioration of IFN-γ and TNF-α-induced intestinal epithelial barrier dysfunction by berberine via suppression of MLCK-MLC phosphorylation signaling pathway

Intestinal mucosal barrier function in health and disease

P304

69-77

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P31

scholarly article

P356

10.7150/IJBS.22259

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P407

English

P433

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P478

http://www.w3.org/2001/XMLSchema#string

P577

2018-01-11T00:00:00Z

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P698

29483826

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P932

5821050

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