Obesity and p16INK4A Downregulation Activate Breast Adipocytes and Promote Their Protumorigenicity.

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Obesity and p16INK4A Downregulation Activate Breast Adipocytes and Promote Their Protumorigenicity.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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Obesity and p16INK4A Downregul ...... omote Their Protumorigenicity.

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Obesity and p16INK4A Downregul ...... omote Their Protumorigenicity.

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Obesity and p16INK4A Downregul ...... omote Their Protumorigenicity.

@en

Obesity and p16INK4A Downregul ...... omote Their Protumorigenicity.

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Obesity and p16INK4A Downregul ...... omote Their Protumorigenicity.

@en

Obesity and p16INK4A Downregul ...... omote Their Protumorigenicity.

@nl

P1476

Obesity and p16INK4A Downregul ...... omote Their Protumorigenicity.

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P2093

Abdelilah Aboussekhra

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Adher Al-Sayed

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Asma Tulbah

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Falah Al-Mohanna

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Huda H Al-Khalaf

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Mrad Amir

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P2860

A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

The epithelial-mesenchymal transition generates cells with properties of stem cells

Adipocytes: impact on tumor growth and potential sites for therapeutic intervention

Adipose tissue as an immunological organ

The Multifaceted Mechanism of Leptin Signaling within Tumor Microenvironment in Driving Breast Cancer Growth and Progression

The p53 cofactor Strap exhibits an unexpected TPR motif and oligonucleotide-binding (OB)-fold structure

Global cancer statistics, 2012

Obesity is associated with macrophage accumulation in adipose tissue

p53, the cellular gatekeeper for growth and division

The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

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scholarly article

P356

10.1128/MCB.00101-17

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English

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P478

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P577

2017-06-19T00:00:00Z

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P698

28630279

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P932

5559683

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