Fructose leads to hepatic steatosis in zebrafish that is reversed by mechanistic target of rapamycin (mTOR) inhibition.
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Preclinical Models for Investigation of Herbal Medicines in Liver Diseases: Update and PerspectiveZebrafish: an important tool for liver disease researchCarbohydrate intake and nonalcoholic fatty liver disease: fructose as a weapon of mass destructionMetabolic insights from zebrafish genetics, physiology, and chemical biologySAK-HV Triggered a Short-period Lipid-lowering Biotherapy Based on the Energy Model of Liver Proliferation via a Novel Pathway.Non-alcoholic fatty liver disease in children: focus on nutritional interventions.A post-developmental genetic screen for zebrafish models of inherited liver disease.Strategies, models and biomarkers in experimental non-alcoholic fatty liver disease researchHigh fat plus high cholesterol diet lead to hepatic steatosis in zebrafish larvae: a novel model for screening anti-hepatic steatosis drugs.MicroRNA expression analysis in high fat diet-induced NAFLD-NASH-HCC progression: study on C57BL/6J mice.Chronic exposure to ethanol causes steatosis and inflammation in zebrafish liver.LITTLE FISH, BIG DATA: ZEBRAFISH AS A MODEL FOR CARDIOVASCULAR AND METABOLIC DISEASE.Dietary fructose as a risk factor for non-alcoholic fatty liver disease (NAFLD).Non-alcoholic fatty liver disease-a chronic disease of the 21(st) century.Hepatic fatty acid biosynthesis is more responsive to protein than carbohydrate in rainbow trout during acute stimulations.Caffeine reduces hepatic lipid accumulation through regulation of lipogenesis and ER stress in zebrafish larvae.Chronically high level of tgfb1a induction causes both hepatocellular carcinoma and cholangiocarcinoma via a dominant Erk pathway in zebrafish.Loss of miR-141/200c ameliorates hepatic steatosis and inflammation by reprogramming multiple signaling pathways in NASH.ATF4 overexpression induces early onset of hyperlipidaemia and hepatic steatosis and enhances adipogenesis in zebrafish.Fructose and glucose regulate mammalian target of rapamycin complex 1 and can activate lipogenic gene expression via distinct pathways.Long-term administration of tacrolimus and everolimus prevents high cholesterol-high fructose-induced steatosis in C57BL/6J mice by inhibiting de-novo lipogenesis.Using zebrafish to model liver diseases-Where do we stand?The Ability of Different Ketohexoses to Alter Apo-A-I Structure and Function In Vitro and to Induce Hepatosteatosis, Oxidative Stress, and Impaired Plasma Lipid Profile in Hyperlipidemic Zebrafish.
P2860
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P2860
Fructose leads to hepatic steatosis in zebrafish that is reversed by mechanistic target of rapamycin (mTOR) inhibition.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
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name
Fructose leads to hepatic stea ...... f rapamycin (mTOR) inhibition.
@en
Fructose leads to hepatic stea ...... echanistic target of rapamycin
@nl
type
label
Fructose leads to hepatic stea ...... f rapamycin (mTOR) inhibition.
@en
Fructose leads to hepatic stea ...... echanistic target of rapamycin
@nl
prefLabel
Fructose leads to hepatic stea ...... f rapamycin (mTOR) inhibition.
@en
Fructose leads to hepatic stea ...... echanistic target of rapamycin
@nl
P2093
P2860
P356
P1433
P1476
Fructose leads to hepatic stea ...... f rapamycin (mTOR) inhibition.
@en
P2093
Leah Gaffney
Randolph P Matthews
Valerie Sapp
P2860
P304
P356
10.1002/HEP.27284
P407
P577
2014-10-01T00:00:00Z