microRNAs expression in endometriosis and their relation to angiogenic factors. - Wikidata - wikimedia - TriplyDB

microRNAs expression in endometriosis and their relation to angiogenic factors.

microRNAs expression in endometriosis and their relation to angiogenic factors.

http://www.wikidata.org/entity/Q50526432

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Endometrial biomarkers for the non-invasive diagnosis of endometriosis Insights into the Regulatory Role of Non-coding RNAs in Cancer Metabolism Systematic enrichment analysis of microRNA expression profiling studies in endometriosis Defining future directions for endometriosis research: workshop report from the 2011 World Congress of Endometriosis In Montpellier, France The Impact of Endometriosis across the Lifespan of Women: Foreseeable Research and Therapeutic Prospects Circulating Micro-RNAs as Diagnostic Biomarkers for Endometriosis: Privation and Promise MicroRNA-16 and microRNA-424 regulate cell-autonomous angiogenic functions in endothelial cells via targeting vascular endothelial growth factor receptor-2 and fibroblast growth factor receptor-1 MicroRNA Profiles in Spontaneous Decidualized Menstrual Endometrium and Early Pregnancy Decidua with Successfully Implanted Embryos Endometrial miR-200c is altered during transformation into cancerous states and targets the expression of ZEBs, VEGFA, FLT1, IKKβ, KLF9, and FBLN5.High-throughput sequencing approach uncovers the miRNome of peritoneal endometriotic lesions and adjacent healthy tissues Peritoneal fluid reduces angiogenesis-related microRNA expression in cell cultures of endometrial and endometriotic tissues from women with endometriosis.Circulating microRNAs as potential biomarkers for endometriosis.MiR-199a attenuates endometrial stromal cell invasiveness through suppression of the IKKβ/NF-κB pathway and reduced interleukin-8 expression.microRNA-21 overexpression contributes to cell proliferation by targeting PTEN in endometrioid endometrial cancer.The Significance of miR-34a Expression in Endometrial Carcinogenesis: Correlation With Expression of p16 and Ki-67 Proteins in Endometrial Cancers.Non-coding RNAs in Uterine Development, Function and Disease.Plasma miR-17-5p, miR-20a and miR-22 are down-regulated in women with endometriosis.MicroRNAs expression profiling of eutopic proliferative endometrium in women with ovarian endometriosis.Influence of ovarian endometrioma on expression of steroid receptor RNA activator, estrogen receptors, vascular endothelial growth factor, and thrombospondin 1 in the surrounding ovarian tissues.New trends for the medical treatment of endometriosis.Genetic, epigenetic and stem cell alterations in endometriosis: new insights and potential therapeutic perspectives.Recent insights on the genetics and epigenetics of endometriosis.MicroRNAs and Endometriosis: Distinguishing Drivers from Passengers in Disease Pathogenesis.miR-196a overexpression activates the MEK/ERK signal and represses the progesterone receptor and decidualization in eutopic endometrium from women with endometriosis.microRNAs related to angiogenesis are dysregulated in endometrioid endometrial cancer.Ginsenoside Rg3 Decreases Fibrotic and Invasive Nature of Endometriosis by Modulating miRNA-27b: In Vitro and In Vivo Studies.Three-Dimensional Power Doppler Vascularization in Women With Ovarian Endometriomas and Relationship With Associated Painful Symptoms.The miRNA Mirage: How Close Are We to Finding a Non-Invasive Diagnostic Biomarker in Endometriosis? A Systematic Review.MicroRNA expression profile in endometriosis: its relation to angiogenesis and fibrinolytic factors.microRNAs and Endometrial Pathophysiology.Ovarian surgery for bilateral endometriomas influences age at menopause.Differential micro ribonucleic acid expression profiling in ovarian endometrioma with leuprolide acetate treatment.Detection of the pan neuronal marker PGP9.5 by immuno-histochemistry and quantitative PCR in eutopic endometrium from women with and without endometriosis.

P2860

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P2860

microRNAs expression in endometriosis and their relation to angiogenic factors.

http://www.wikidata.org/entity/Q50526432

description

2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

@zh-hans

2011年学术文章

@zh-my

2011年学术文章

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2011年學術文章

@yue

2011年學術文章

@zh-hant

name

microRNAs expression in endometriosis and their relation to angiogenic factors.

@en

microRNAs expression in endometriosis and their relation to angiogenic factors.

@nl

type

label

microRNAs expression in endometriosis and their relation to angiogenic factors.

@en

microRNAs expression in endometriosis and their relation to angiogenic factors.

@nl

prefLabel

microRNAs expression in endometriosis and their relation to angiogenic factors.

@en

microRNAs expression in endometriosis and their relation to angiogenic factors.

@nl

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P356

P1433

Human Reproduction

P1476

microRNAs expression in endometriosis and their relation to angiogenic factors

@en

P2093

Amparo Estellés

http://www.w3.org/2001/XMLSchema#string

Juan Gilabert

http://www.w3.org/2001/XMLSchema#string

Juan Gilabert-Estellés

http://www.w3.org/2001/XMLSchema#string

Luis A Ramón

http://www.w3.org/2001/XMLSchema#string

Melitina Chirivella

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P2860

miRiad roles for the miR-17-92 cluster in development and disease

Circulating microRNAs as stable blood-based markers for cancer detection.

MicroRNAs: genomics, biogenesis, mechanism, and function

Members of the microRNA-17-92 cluster exhibit a cell-intrinsic antiangiogenic function in endothelial cells

Insights into the role of microRNAs in cardiac diseases: from biological signalling to therapeutic targets

MicroRNA signature and regulatory functions in the endometrium during normal and disease states

Angiogenesis in health and disease

Augmentation of tumor angiogenesis by a Myc-activated microRNA cluster

MicroRNA-regulated pathways associated with endometriosis.

Genomic profiling of microRNAs and messenger RNAs reveals hormonal regulation in microRNA expression in human endometrium

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1082-1090

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10.1093/HUMREP/DER025

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5

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26

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Francisco España

Aitana Braza-Boïls

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2011-02-18T00:00:00Z

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49849656

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21335415

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