Rescue of the abnormal skeletal phenotype in Ts65Dn Down syndrome mice using genetic and therapeutic modulation of trisomic Dyrk1a.
about
Differential effects of Epigallocatechin-3-gallate containing supplements on correcting skeletal defects in a Down syndrome mouse model.Influence of prenatal EGCG treatment and Dyrk1a dosage reduction on craniofacial features associated with Down syndrome.Trans-acting epigenetic effects of chromosomal aneuploidies: lessons from Down syndrome and mouse models.Targeting trisomic treatments: optimizing Dyrk1a inhibition to improve Down syndrome deficits.Epigallocatechin-3-gallate (EGCG) consumption in the Ts65Dn model of Down syndrome fails to improve behavioral deficits and is detrimental to skeletal phenotypes.Sclerostin Antibody Treatment Stimulates Bone Formation to Normalize Bone Mass in Male Down Syndrome Mice
P2860
Rescue of the abnormal skeletal phenotype in Ts65Dn Down syndrome mice using genetic and therapeutic modulation of trisomic Dyrk1a.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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name
Rescue of the abnormal skeleta ...... modulation of trisomic Dyrk1a.
@en
Rescue of the abnormal skeleta ...... modulation of trisomic Dyrk1a.
@nl
type
label
Rescue of the abnormal skeleta ...... modulation of trisomic Dyrk1a.
@en
Rescue of the abnormal skeleta ...... modulation of trisomic Dyrk1a.
@nl
prefLabel
Rescue of the abnormal skeleta ...... modulation of trisomic Dyrk1a.
@en
Rescue of the abnormal skeleta ...... modulation of trisomic Dyrk1a.
@nl
P2093
P2860
P356
P1476
Rescue of the abnormal skeleta ...... modulation of trisomic Dyrk1a.
@en
P2093
Irushi Abeysekera
Jiliang Li
Joshua D Blazek
P2860
P304
P356
10.1093/HMG/DDV284
P577
2015-07-23T00:00:00Z