Poor memory B cell generation contributes to non-protective responses to DTaP vaccine antigens in otitis-prone children.

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2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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Poor memory B cell generation ...... gens in otitis-prone children.

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Poor memory B cell generation ...... gens in otitis-prone children.

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Item

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Poor memory B cell generation ...... gens in otitis-prone children.

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Poor memory B cell generation ...... gens in otitis-prone children.

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Poor memory B cell generation ...... gens in otitis-prone children.

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Poor memory B cell generation ...... gens in otitis-prone children.

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P1476

Poor memory B cell generation ...... gens in otitis-prone children.

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P2093

M E Pichichero

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S Basha

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P2860

Search for correlates of protective immunity conferred by anthrax vaccine.

The TLR9 ligand CpG promotes the acquisition of Plasmodium falciparum-specific memory B cells in malaria-naive individuals.

Differential impact of respiratory syncytial virus and parainfluenza virus on the frequency of acute otitis media is explained by lower adaptive and innate immune responses in otitis-prone children.

Immunological memory in humans.

HIV-1 envelope induces memory B cell responses that correlate with plasma antibody levels after envelope gp120 protein vaccination or HIV-1 infection.

Reduced memory CD4+ T-cell generation in the circulation of young children may contribute to the otitis-prone condition.

Quantitation of rare memory B cell populations by two independent and complementary approaches

Serum antibody response to five Streptococcus pneumoniae proteins during acute otitis media in otitis-prone and non-otitis-prone children

Reduced serum IgG responses to pneumococcal antigens in otitis-prone children may be due to poor memory B-cell generation.

Vaccine candidates PhtD and PhtE of Streptococcus pneumoniae are adhesins that elicit functional antibodies in humans.

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314-322

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scholarly article

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10.1111/CEI.12660

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182

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2015-07-03T00:00:00Z

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26138025

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4636893

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