Epithelial IGF1R is dispensable for IGF2 mediated enhanced intestinal adaptation in retinoblastoma-deficient mice.

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Epithelial IGF1R is dispensable for IGF2 mediated enhanced intestinal adaptation in retinoblastoma-deficient mice.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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Epithelial IGF1R is dispensabl ...... retinoblastoma-deficient mice.

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Epithelial IGF1R is dispensabl ...... retinoblastoma-deficient mice.

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Epithelial IGF1R is dispensabl ...... retinoblastoma-deficient mice.

@en

Epithelial IGF1R is dispensabl ...... retinoblastoma-deficient mice.

@nl

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Epithelial IGF1R is dispensabl ...... retinoblastoma-deficient mice.

@en

Epithelial IGF1R is dispensabl ...... retinoblastoma-deficient mice.

@nl

P1476

Epithelial IGF1R is dispensabl ...... retinoblastoma-deficient mice.

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Brad W Warner

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Christopher R Erwin

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Jose L Diaz-Miron

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Joshua Sommovilla

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Jun Guo

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Pamela M Choi

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Raphael C Sun

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P2860

Disruption of retinoblastoma protein expression in the intestinal epithelium impairs lipid absorption

Insulin-like growth factor 2 and its enterocyte receptor are not required for adaptation in response to massive small bowel resection.

IGF-2 is necessary for retinoblastoma-mediated enhanced adaptation after small-bowel resection

Both epidermal growth factor and insulin-like growth factor receptors are dispensable for structural intestinal adaptation.

Causes and management of intestinal failure in children.

Mechanisms of intestinal adaptation.

Regulation of retinoblastoma protein (Rb) by p21 is critical for adaptation to massive small bowel resection.

IGF-2 mediates intestinal mucosal hyperplasia in retinoblastoma protein (Rb)-deficient mice.

Insulin-like growth factor 1: common mediator of multiple enterotrophic hormones and growth factors.

Molecular basis of intestinal adaptation: the role of the insulin-like growth factor system.

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1026-1030

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scholarly article

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10.1016/J.JPEDSURG.2017.03.030

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English

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P478

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P577

2017-03-18T00:00:00Z

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P698

28343662

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P921

retinoblastoma

P932

5466888

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