Kv3.1-containing K(+) channels are reduced in untreated schizophrenia and normalized with antipsychotic drugs.
about
The impact of NMDA receptor hypofunction on GABAergic neurons in the pathophysiology of schizophreniaIdentification of Distinct Psychosis Biotypes Using Brain-Based BiomarkersMolecular substrates of schizophrenia: homeostatic signaling to connectivityThe translationally relevant mouse model of the 15q13.3 microdeletion syndrome reveals deficits in neuronal spike firing matching clinical neurophysiological biomarkers seen in schizophrenia.Oxidative stress-driven parvalbumin interneuron impairment as a common mechanism in models of schizophrenia.Early developmental disturbances of cortical inhibitory neurons: contribution to cognitive deficits in schizophrenia.Redox dysregulation, neuroinflammation, and NMDA receptor hypofunction: A "central hub" in schizophrenia pathophysiology?Alterations in cortical network oscillations and parvalbumin neurons in schizophrenia.Perineuronal nets and schizophrenia: the importance of neuronal coatings.AMIGO-Kv2.1 Potassium Channel Complex Is Associated With Schizophrenia-Related Phenotypes.A Role for the Transcription Factor Nk2 Homeobox 1 in Schizophrenia: Convergent Evidence from Animal and Human StudiesLower gene expression for KCNS3 potassium channel subunit in parvalbumin-containing neurons in the prefrontal cortex in schizophrenia.Psychosis subgroups differ in intrinsic neural activity but not task-specific processing.Altering the course of schizophrenia: progress and perspectives.Myoclonus epilepsy and ataxia due to KCNC1 mutation: Analysis of 20 cases and K+ channel properties.Potassium channel gene associations with joint processing speed and white matter impairments in schizophrenia.Distinct Physiological Maturation of Parvalbumin-Positive Neuron Subtypes in Mouse Prefrontal Cortex.Antimanic Efficacy of a Novel Kv3 Potassium Channel Modulator.Kv3 Channels: Enablers of Rapid Firing, Neurotransmitter Release, and Neuronal Endurance.Parvalbumin fast-spiking interneurons are selectively altered by pediatric traumatic brain injury.Identification and characterization of site-specific N-glycosylation in the potassium channel Kv3.1b.Loss of Function of KCNC1 is associated with intellectual disability without seizures.Multidimensional Connectomics and Treatment-Resistant Schizophrenia: Linking Phenotypic Circuits to Targeted Therapeutics
P2860
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P2860
Kv3.1-containing K(+) channels are reduced in untreated schizophrenia and normalized with antipsychotic drugs.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh-hant
name
Kv3.1-containing K
@nl
Kv3.1-containing K(+) channels ...... ized with antipsychotic drugs.
@en
type
label
Kv3.1-containing K
@nl
Kv3.1-containing K(+) channels ...... ized with antipsychotic drugs.
@en
prefLabel
Kv3.1-containing K
@nl
Kv3.1-containing K(+) channels ...... ized with antipsychotic drugs.
@en
P2093
P2860
P356
P1433
P1476
Kv3.1-containing K(+) channels ...... ized with antipsychotic drugs.
@en
P2093
P2860
P2888
P304
P356
10.1038/MP.2013.49
P407
P577
2013-04-30T00:00:00Z