MiR-21 is overexpressed in response to high glucose and protects endothelial cells from apoptosis.
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Interplay Between Metabolism and Oncogenic Process: Role of microRNAsThe Role of MicroRNAs in Diabetic Complications-Special Emphasis on Wound HealingSmall molecules, big effects: the role of microRNAs in regulation of cardiomyocyte deathConsumption of distinct dietary lipids during early pregnancy differentially modulates the expression of microRNAs in mothers and offspringIntegrated analysis of microarray data of atherosclerotic plaques: modulation of the ubiquitin-proteasome system.miR-182 aids in receptive endometrium development in dairy goats by down-regulating PTN expressionMicroRNA-26a prevents endothelial cell apoptosis by directly targeting TRPC6 in the setting of atherosclerosis.MiR-30b Is Involved in the Homocysteine-Induced Apoptosis in Human Coronary Artery Endothelial Cells by Regulating the Expression of Caspase 3MiR-21-5p and miR-126a-3p levels in plasma and circulating angiogenic cells: relationship with type 2 diabetes complications.Circulating miRNAs as intercellular messengers, potential biomarkers and therapeutic targets for Type 2 diabetes.Epigenetics and Cardiovascular Disease in Diabetes.Biomarkers of Diabetic Retinopathy.Modulation of the vascular endothelium functioning by dietary components, the role of epigenetics.Emerging Roles for MicroRNAs in Diabetic Microvascular Disease: Novel Targets for Therapy.Long noncoding RNA MEG3 suppressed endothelial cell proliferation and migration through regulating miR-21Down regulation of Jag-1 in VSMCs contributes to impaired angiogenesis under high glucose condition: Experimental study using aortic rings of rats.The MicroRNA-21 signaling pathway is involved in prorenin receptor (PRR) -induced VEGF expression in ARPE-19 cells under a hyperglycemic condition.Screening differential miRNAs responsible for permeability increase in HUVECs infected with influenza A virus.Roles of miRNAs and long noncoding RNAs in the progression of diabetic retinopathy.The microRNA miR-124 inhibits vascular smooth muscle cell proliferation by targeting S100 calcium-binding protein A4 (S100A4).Acidic preconditioning of endothelial colony-forming cells (ECFC) promote vasculogenesis under proinflammatory and high glucose conditions in vitro and in vivo.
P2860
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P2860
MiR-21 is overexpressed in response to high glucose and protects endothelial cells from apoptosis.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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name
MiR-21 is overexpressed in res ...... othelial cells from apoptosis.
@en
MiR-21 is overexpressed in res ...... othelial cells from apoptosis.
@nl
type
label
MiR-21 is overexpressed in res ...... othelial cells from apoptosis.
@en
MiR-21 is overexpressed in res ...... othelial cells from apoptosis.
@nl
prefLabel
MiR-21 is overexpressed in res ...... othelial cells from apoptosis.
@en
MiR-21 is overexpressed in res ...... othelial cells from apoptosis.
@nl
P2093
P356
P1476
MiR-21 is overexpressed in res ...... othelial cells from apoptosis.
@en
P2093
P304
P356
10.1055/S-0033-1345169
P577
2013-06-14T00:00:00Z