CSL regulates AKT to mediate androgen independence in prostate cancer progression.
about
Activation of AKT negatively regulates the pro-apoptotic function of death-associated protein kinase 3 (DAPK3) in prostate cancer.Dissecting the roles of the androgen receptor in prostate cancer from molecular perspectives.The Metabolic Phenotype of Prostate Cancer.Transdermal administration of melatonin coupled to cryopass laser treatment as noninvasive therapy for prostate cancer.Activation of Notch pathway is linked with epithelial-mesenchymal transition in prostate cancer cells.
P2860
CSL regulates AKT to mediate androgen independence in prostate cancer progression.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年學術文章
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2015年學術文章
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name
CSL regulates AKT to mediate androgen independence in prostate cancer progression.
@en
CSL regulates AKT to mediate androgen independence in prostate cancer progression.
@nl
type
label
CSL regulates AKT to mediate androgen independence in prostate cancer progression.
@en
CSL regulates AKT to mediate androgen independence in prostate cancer progression.
@nl
prefLabel
CSL regulates AKT to mediate androgen independence in prostate cancer progression.
@en
CSL regulates AKT to mediate androgen independence in prostate cancer progression.
@nl
P2093
P2860
P356
P1433
P1476
CSL regulates AKT to mediate androgen independence in prostate cancer progression.
@en
P2093
Huiyan Wang
Lixia Zhang
Yong Jiang
P2860
P304
P356
10.1002/PROS.23104
P577
2015-10-06T00:00:00Z