Akap-mediated signalling: the importance of being in the right place at the right time.

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Akap-mediated signalling: the importance of being in the right place at the right time.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

@wuu

2016年学术文章

@zh

2016年学术文章

@zh-cn

2016年学术文章

@zh-hans

2016年学术文章

@zh-my

2016年学术文章

@zh-sg

2016年學術文章

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2016年學術文章

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Akap-mediated signalling: the importance of being in the right place at the right time.

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Akap-mediated signalling: the importance of being in the right place at the right time.

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Item

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Akap-mediated signalling: the importance of being in the right place at the right time.

@en

Akap-mediated signalling: the importance of being in the right place at the right time.

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Akap-mediated signalling: the importance of being in the right place at the right time.

@en

Akap-mediated signalling: the importance of being in the right place at the right time.

@nl

P1476

Akap-mediated signalling: the importance of being in the right place at the right time.

@en

P2093

Cinzia Perrino

http://www.w3.org/2001/XMLSchema#string

P2860

PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts

Akap1 Deficiency Promotes Mitochondrial Aberrations and Exacerbates Cardiac Injury Following Permanent Coronary Ligation via Enhanced Mitophagy and Apoptosis

Membrane-bound protein kinase A inhibits smooth muscle cell proliferation in vitro and in vivo by amplifying cAMP-protein kinase A signals

AKAP79/150 anchoring of calcineurin controls neuronal L-type Ca2+ channel activity and nuclear signaling

AKAP150-dependent cooperative TRPV4 channel gating is central to endothelium-dependent vasodilation and is disrupted in hypertension.

Seven-transmembrane-spanning receptors and heart function.

Carvedilol reverses cardiac insufficiency in AKAP5 knockout mice by normalizing the activities of calcineurin and CaMKII.

Reversal of cardiac remodeling by modulation of adrenergic receptors: a new frontier in heart failure.

AKAP150 participates in calcineurin/NFAT activation during the down-regulation of voltage-gated K(+) currents in ventricular myocytes following myocardial infarction.

AKAP signaling complexes: pointing towards the next generation of therapeutic targets?

P304

115-117

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P31

scholarly article

editorial

P356

10.1093/CVR/CVW254

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

113

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P50

Bruno Trimarco

P577

2016-12-21T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28003270

http://www.w3.org/2001/XMLSchema#string