Endoplasmic Reticulum Stress-Induced CHOP Inhibits PGC-1α and Causes Mitochondrial Dysfunction in Diabetic Embryopathy.

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The current status and future of cardiac stem/progenitor cell therapy for congenital heart defects from diabetic pregnancy.

P2860

Q46302230-3E444846-5A48-417E-8DCC-2F5A1526811B

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Endoplasmic Reticulum Stress-Induced CHOP Inhibits PGC-1α and Causes Mitochondrial Dysfunction in Diabetic Embryopathy.

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http://www.wikidata.org/entity/Q51015129

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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name

Endoplasmic Reticulum Stress-I ...... ction in Diabetic Embryopathy.

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Endoplasmic Reticulum Stress-I ...... ction in Diabetic Embryopathy.

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type

Item

label

Endoplasmic Reticulum Stress-I ...... ction in Diabetic Embryopathy.

@en

Endoplasmic Reticulum Stress-I ...... ction in Diabetic Embryopathy.

@nl

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Endoplasmic Reticulum Stress-I ...... ction in Diabetic Embryopathy.

@en

Endoplasmic Reticulum Stress-I ...... ction in Diabetic Embryopathy.

@nl

P1476

Endoplasmic Reticulum Stress-I ...... nction in Diabetic Embryopathy

@en

P2093

Gentao Liu

http://www.w3.org/2001/XMLSchema#string

Jianxiang Zhong

http://www.w3.org/2001/XMLSchema#string

Peixin Yang

http://www.w3.org/2001/XMLSchema#string

Xi Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Critical and functional regulation of CHOP (C/EBP homologous protein) through the N-terminal portion

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Birth defects in pregestational diabetes: Defect range, glycemic threshold and pathogenesis

ER stress-induced cell death mechanisms

CHOP, a novel developmentally regulated nuclear protein that dimerizes with transcription factors C/EBP and LAP and functions as a dominant-negative inhibitor of gene transcription

Multipotent neural cell lines can engraft and participate in development of mouse cerebellum

Defects in adaptive energy metabolism with CNS-linked hyperactivity in PGC-1alpha null mice

Protein kinase C-alpha suppresses autophagy and induces neural tube defects via miR-129-2 in diabetic pregnancy.

Metabolic control of mitochondrial biogenesis through the PGC-1 family regulatory network

Ask1 gene deletion blocks maternal diabetes-induced endoplasmic reticulum stress in the developing embryo by disrupting the unfolded protein response signalosome

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275-285

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scholarly article

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10.1093/TOXSCI/KFX096

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P433

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158

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P50

Daoyin Dong

P577

2017-08-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

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28482072

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P921

endoplasmic reticulum

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5837255

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Endoplasmic Reticulum Stress-Induced CHOP Inhibits PGC-1α and Causes Mitochondrial Dysfunction in Diabetic Embryopathy.

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P2860

P2860

Endoplasmic Reticulum Stress-Induced CHOP Inhibits PGC-1α and Causes Mitochondrial Dysfunction in Diabetic Embryopathy.

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type

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P1433

P1476

P2093

P2860

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P31

P356

P433

P478

P50

P577

P698

P921

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P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

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P921

P932