Epac activation inhibits IL-6-induced cardiac myocyte dysfunction.

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Epac activation inhibits IL-6-induced cardiac myocyte dysfunction.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

@zh-cn

2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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name

Epac activation inhibits IL-6-induced cardiac myocyte dysfunction.

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Epac activation inhibits IL-6-induced cardiac myocyte dysfunction.

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Item

label

Epac activation inhibits IL-6-induced cardiac myocyte dysfunction.

@en

Epac activation inhibits IL-6-induced cardiac myocyte dysfunction.

@nl

prefLabel

Epac activation inhibits IL-6-induced cardiac myocyte dysfunction.

@en

Epac activation inhibits IL-6-induced cardiac myocyte dysfunction.

@nl

P1476

Epac activation inhibits IL-6-induced cardiac myocyte dysfunction

@en

P2093

Chen Liang

http://www.w3.org/2001/XMLSchema#string

Huiling Jin

http://www.w3.org/2001/XMLSchema#string

Kenji Suita

http://www.w3.org/2001/XMLSchema#string

Masanari Umemura

http://www.w3.org/2001/XMLSchema#string

Meihua Jin

http://www.w3.org/2001/XMLSchema#string

Motohiko Sato

http://www.w3.org/2001/XMLSchema#string

Rajesh Prajapati

http://www.w3.org/2001/XMLSchema#string

Reiko Kurotani

http://www.w3.org/2001/XMLSchema#string

Satoshi Okumura

http://www.w3.org/2001/XMLSchema#string

Takayuki Fujita

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P2860

Epac is a Rap1 guanine-nucleotide-exchange factor directly activated by cyclic AMP

A family of cAMP-binding proteins that directly activate Rap1

The protein kinase A anchoring protein mAKAP coordinates two integrated cAMP effector pathways.

Epac-selective cAMP analog 8-pCPT-2'-O-Me-cAMP as a stimulus for Ca2+-induced Ca2+ release and exocytosis in pancreatic beta-cells.

JAK2/STAT3, not ERK1/2, mediates interleukin-6-induced activation of inducible nitric-oxide synthase and decrease in contractility of adult ventricular myocytes

Prostaglandin E2-activated Epac promotes neointimal formation of the rat ductus arteriosus by a process distinct from that of cAMP-dependent protein kinase A

Epac mediates beta-adrenergic receptor-induced cardiomyocyte hypertrophy

Exchange protein activated by cyclic AMP (Epac)-mediated induction of suppressor of cytokine signaling 3 (SOCS-3) in vascular endothelial cells.

Endotoxin-induced myocardial dysfunction: effects of macrophage migration inhibitory factor neutralization.

Elevated cyclic AMP and PDE4 inhibition induce chemokine expression in human monocyte-derived macrophages

P2888

s12576-016-0509-5

P304

77-87

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P31

scholarly article

P356

10.1007/S12576-016-0509-5

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2016-12-19T00:00:00Z

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P698

27995459

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P932

6353818

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