GPR4 deficiency alleviates intestinal inflammation in a mouse model of acute experimental colitis.

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Acidosis Activates Endoplasmic Reticulum Stress Pathways through GPR4 in Human Vascular Endothelial Cells.The proton-activated receptor GPR4 modulates intestinal inflammation.

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GPR4 deficiency alleviates intestinal inflammation in a mouse model of acute experimental colitis.

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http://www.wikidata.org/entity/Q51274483

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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name

GPR4 deficiency alleviates int ...... of acute experimental colitis.

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GPR4 deficiency alleviates int ...... of acute experimental colitis.

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GPR4 deficiency alleviates int ...... of acute experimental colitis.

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GPR4 deficiency alleviates int ...... of acute experimental colitis.

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GPR4 deficiency alleviates int ...... of acute experimental colitis.

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GPR4 deficiency alleviates int ...... of acute experimental colitis.

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P1476

GPR4 deficiency alleviates int ...... of acute experimental colitis.

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P2093

Calvin R Justus

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Dorcas O'Rourke

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Edward J Sanderlin

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Elizabeth A Krewson

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Heng Hong

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Joani Zary Oswald

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Kvin Lertpiriyapong

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Nancy R Leffler

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Qi Cai

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Vasudevan Bakthavatchalu

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P2860

Apolipoprotein A-IV inhibits experimental colitis

Cloning, sequencing and tissue distribution of two related G protein-coupled receptor candidates expressed prominently in human lung tissue

Isolation of a novel G protein-coupled receptor (GPR4) localized to chromosome 19q13.3

Identification of T cell death-associated gene 8 (TDAG8) as a novel acid sensing G-protein-coupled receptor

Differential proton sensitivity of related G protein-coupled receptors T cell death-associated gene 8 and G2A expressed in immune cells

Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci

Vedolizumab for the treatment of ulcerative colitis and Crohn's disease

Science review: extracellular acidosis and the immune response: clinical and physiologic implications

Progressive multifocal leukoencephalopathy: an unexpected complication of modern therapeutic monoclonal antibody therapies

Acidosis activation of the proton-sensing GPR4 receptor stimulates vascular endothelial cell inflammatory responses revealed by transcriptome analysis

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569-584

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scholarly article

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10.1016/J.BBADIS.2016.12.005

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1863

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Li V. Yang

James G. Fox

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2016-12-07T00:00:00Z

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27940273

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5222690

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GPR4 deficiency alleviates intestinal inflammation in a mouse model of acute experimental colitis.

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P2860

P2860

GPR4 deficiency alleviates intestinal inflammation in a mouse model of acute experimental colitis.

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P1476

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