Nuclear galectin-1-FOXP3 interaction dampens the tumor-suppressive properties of FOXP3 in breast cancer.

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Nuclear galectin-1-FOXP3 interaction dampens the tumor-suppressive properties of FOXP3 in breast cancer.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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Nuclear galectin-1-FOXP3 inter ...... ies of FOXP3 in breast cancer.

@en

Nuclear galectin-1-FOXP3 inter ...... ies of FOXP3 in breast cancer.

@nl

type

Item

label

Nuclear galectin-1-FOXP3 inter ...... ies of FOXP3 in breast cancer.

@en

Nuclear galectin-1-FOXP3 inter ...... ies of FOXP3 in breast cancer.

@nl

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Nuclear galectin-1-FOXP3 inter ...... ies of FOXP3 in breast cancer.

@en

Nuclear galectin-1-FOXP3 inter ...... ies of FOXP3 in breast cancer.

@nl

P1476

Nuclear galectin-1-FOXP3 inter ...... ties of FOXP3 in breast cancer

@en

P2093

Cheng Zeng

http://www.w3.org/2001/XMLSchema#string

Cun Zhang

http://www.w3.org/2001/XMLSchema#string

Dong Fan

http://www.w3.org/2001/XMLSchema#string

Huadong Zhao

http://www.w3.org/2001/XMLSchema#string

Kuo Zhang

http://www.w3.org/2001/XMLSchema#string

Meng Li

http://www.w3.org/2001/XMLSchema#string

Qiang Hao

http://www.w3.org/2001/XMLSchema#string

Shuning Wang

http://www.w3.org/2001/XMLSchema#string

Wangqian Zhang

http://www.w3.org/2001/XMLSchema#string

Wei Zhang

http://www.w3.org/2001/XMLSchema#string

P2860

Analysis of FOXP3 reveals multiple domains required for its function as a transcriptional repressor

Crucial role of FOXP3 in the development and function of human CD25+CD4+ regulatory T cells

FOXP3 up-regulates p21 expression by site-specific inhibition of histone deacetylase 2/histone deacetylase 4 association to the locus

Galectin-1 is a stromal cell ligand of the pre-B cell receptor (BCR) implicated in synapse formation between pre-B and stromal cells and in pre-BCR triggering.

The Role of Galectin-1 in Cancer Progression, and Synthetic Multivalent Systems for the Study of Galectin-1

Loss of ERα induces amoeboid-like migration of breast cancer cells by downregulating vinculin.

Proteomic profiling identifies breast tumor metastasis-associated factors in an isogenic model

Foxp3+ regulatory T cells maintain the bone marrow microenvironment for B cell lymphopoiesis.

Distinct EMT programs control normal mammary stem cells and tumour-initiating cells.

Acidity generated by the tumor microenvironment drives local invasion

P304

416

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scholarly article

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10.1038/S41419-018-0448-6

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P478

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P50

Xiaochang Xue

P577

2018-04-01T00:00:00Z

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29549328

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5856744

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