Nociceptive-specific activation of ERK in spinal neurons contributes to pain hypersensitivity.
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Biochanin A, a naturally occurring inhibitor of fatty acid amide hydrolaseResolvins RvE1 and RvD1 attenuate inflammatory pain via central and peripheral actionsNeuronal circuitry for pain processing in the dorsal hornHSV-mediated expression of interleukin-4 in dorsal root ganglion neurons reduces neuropathic pain.Impaired inflammatory pain and thermal hyperalgesia in mice expressing neuron-specific dominant negative mitogen activated protein kinase kinase (MEK)Biological Roles of Resolvins and Related Substances in the Resolution of PainThe glutamatergic system as a target for neuropathic pain reliefWhole-brain activity mapping onto a zebrafish brain atlas.Orofacial Neuropathic Pain Leads to a Hyporesponsive Barrel Cortex with Enhanced Structural Synaptic PlasticityEmerging targets in neuroinflammation-driven chronic painEmerging roles of resolvins in the resolution of inflammation and painAβ40 modulates GABA(A) receptor α6 subunit expression and rat cerebellar granule neuron maturation through the ERK/mTOR pathwayDifferential activation of p38 and extracellular signal-regulated kinase in spinal cord in a model of bee venom-induced inflammation and hyperalgesiaFrequency-dependent ERK phosphorylation in spinal neurons by electric stimulation of the sciatic nerve and the role in electrophysiological activityActivation of the neuronal extracellular signal-regulated kinase 2 in the spinal cord dorsal horn is required for complete Freund's adjuvant-induced pain hypersensitivityMAP kinase and pain.Activation of extracellular signal-regulated kinase in the anterior cingulate cortex contributes to the induction and expression of affective pain.Genetic targeting of ERK1 suggests a predominant role for ERK2 in murine pain modelsChronic itch development in sensory neurons requires BRAF signaling pathways.Peripheral metabotropic glutamate receptors as drug targets for pain relief.The mitogen and stress-activated protein kinase 1 regulates the rapid epigenetic tagging of dorsal horn neurons and nocifensive behaviourRegion- or state-related differences in expression and activation of extracellular signal-regulated kinases (ERKs) in naïve and pain-experiencing rats.Extracellular signal-regulated kinase (ERK) and protein kinase B (AKT) pathways involved in spinal cord stimulation (SCS)-induced vasodilation.Metabotropic glutamate receptors: novel targets for pain relief.Effects of estrogens and bladder inflammation on mitogen-activated protein kinases in lumbosacral dorsal root ganglia from adult female ratsAstroglia in medullary dorsal horn (trigeminal spinal subnucleus caudalis) are involved in trigeminal neuropathic pain mechanisms.Chronic inflammation and estradiol interact through MAPK activation to affect TMJ nociceptive processing by trigeminal caudalis neurons.The chemerin receptor 23 agonist, chemerin, attenuates monosynaptic C-fibre input to lamina I neurokinin 1 receptor expressing rat spinal cord neurons in inflammatory painChronic tooth pulp inflammation induces persistent expression of phosphorylated ERK (pERK) and phosphorylated p38 (pp38) in trigeminal subnucleus caudalis.Targeting the Microglial Signaling Pathways: New Insights in the Modulation of Neuropathic Pain.N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain.Expression genetics identifies spinal mechanisms supporting formalin late phase behaviorsIncreased spinal prodynorphin gene expression in reinflammation-associated hyperalgesia after neonatal inflammatory insultRole of the oestrogen receptors GPR30 and ERalpha in peripheral sensitization: relevance to trigeminal pain disorders in women.Lesion of the dopaminergic nigrostriatal pathway induces trigeminal dynamic mechanical allodyniaβ-arrestin-2 regulates NMDA receptor function in spinal lamina II neurons and duration of persistent pain.Gp120 in the pathogenesis of human immunodeficiency virus-associated pain.Spinal inhibition of p38 MAP kinase reduces inflammatory and neuropathic pain in male but not female mice: Sex-dependent microglial signaling in the spinal cord.[EXPRESS] CXCL12/CXCR4 signaling mediated ERK1/2 activation in spinal cord contributes to the pathogenesis of postsurgical pain in rats.Pathobiology of visceral pain: molecular mechanisms and therapeutic implications. III. Visceral afferent pathways: a source of new therapeutic targets for abdominal pain.
P2860
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P2860
Nociceptive-specific activation of ERK in spinal neurons contributes to pain hypersensitivity.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh-hant
name
Nociceptive-specific activatio ...... utes to pain hypersensitivity.
@en
Nociceptive-specific activatio ...... utes to pain hypersensitivity.
@nl
type
label
Nociceptive-specific activatio ...... utes to pain hypersensitivity.
@en
Nociceptive-specific activatio ...... utes to pain hypersensitivity.
@nl
prefLabel
Nociceptive-specific activatio ...... utes to pain hypersensitivity.
@en
Nociceptive-specific activatio ...... utes to pain hypersensitivity.
@nl
P2093
P2860
P356
P1433
P1476
Nociceptive-specific activatio ...... utes to pain hypersensitivity.
@en
P2093
P2860
P2888
P304
P356
10.1038/16040
P407
P50
P577
1999-12-01T00:00:00Z