Tenascin-C Is a Major Component of the Fibrogenic Niche in Kidney Fibrosis.
about
Unilateral nephrectomy diminishes ischemic acute kidney injury through enhanced perfusion and reduced pro-inflammatory and pro-fibrotic responses.In Vivo siRNA Delivery and Rebound of Renal LRP2 in Mice.Fibroblast-Specific β-catenin Signaling Dictates the Outcome of AKI.In wound repair vimentin mediates the transition of mesenchymal leader cells to a myofibroblast phenotype.
P2860
Tenascin-C Is a Major Component of the Fibrogenic Niche in Kidney Fibrosis.
description
2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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name
Tenascin-C Is a Major Component of the Fibrogenic Niche in Kidney Fibrosis.
@en
Tenascin-C Is a Major Component of the Fibrogenic Niche in Kidney Fibrosis.
@nl
type
label
Tenascin-C Is a Major Component of the Fibrogenic Niche in Kidney Fibrosis.
@en
Tenascin-C Is a Major Component of the Fibrogenic Niche in Kidney Fibrosis.
@nl
prefLabel
Tenascin-C Is a Major Component of the Fibrogenic Niche in Kidney Fibrosis.
@en
Tenascin-C Is a Major Component of the Fibrogenic Niche in Kidney Fibrosis.
@nl
P2093
P2860
P356
P1476
Tenascin-C Is a Major Component of the Fibrogenic Niche in Kidney Fibrosis.
@en
P2093
Donna B Stolz
Roderick J Tan
Youhua Liu
P2860
P304
P356
10.1681/ASN.2016020165
P577
2016-09-09T00:00:00Z