Uteroplacental insufficiency programs regional vascular dysfunction and alters arterial stiffness in female offspring. - Wikidata - wikimedia - TriplyDB

Uteroplacental insufficiency programs regional vascular dysfunction and alters arterial stiffness in female offspring.

Uteroplacental insufficiency programs regional vascular dysfunction and alters arterial stiffness in female offspring.

http://www.wikidata.org/entity/Q51570553

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The Programming Power of the Placenta Leptin in pregnancy and development: a contributor to adulthood disease?Pregnancy as a critical window for blood pressure regulation in mother and child: programming and reprogramming.Cardiovascular function in adulthood following intrauterine growth restriction with abnormal fetal blood flow.Prenatal exposure to dexamethasone in the mouse alters cardiac growth patterns and increases pulse pressure in aged male offspring Estrogen receptor subtypes mediate distinct microvascular dilation and reduction in [Ca2+]I in mesenteric microvessels of female rat.Low birth weight is strongly associated with the risk of deep infiltrating endometriosis: results of a 743 case-control study.Alterations in maternal and fetal heart functions accompany failed spiral arterial remodeling in pregnant mice.Pre-eclampsia and offspring cardiovascular health: mechanistic insights from experimental studies.Normal lactational environment restores cardiomyocyte number after uteroplacental insufficiency: implications for the preterm neonate Increased MMPs expression and decreased contraction in the rat myometrium during pregnancy and in response to prolonged stretch and sex hormones.Uteroplacental insufficiency leads to hypertension, but not glucose intolerance or impaired skeletal muscle mitochondrial biogenesis, in 12-month-old rats.Relaxin mediates uterine artery compliance during pregnancy and increases uterine blood flow.Adaptive increases in expression and vasodilator activity of estrogen receptor subtypes in a blood vessel-specific pattern during pregnancy.Sex differences in the enhanced responsiveness to acute angiotensin II in growth-restricted rats: role of fasudil, a Rho kinase inhibitor.Subtype-specific estrogen receptor-mediated vasodilator activity in the cephalic, thoracic, and abdominal vasculature of female rat.Pregnancy-associated adaptations in [Ca2+]i-dependent and Ca2+ sensitization mechanisms of venous contraction: implications in pregnancy-related venous disorders.EMMPRIN-mediated induction of uterine and vascular matrix metalloproteinases during pregnancy and in response to estrogen and progesterone Enhanced endothelin receptor type B-mediated vasodilation and underlying [Ca²⁺]i in mesenteric microvessels of pregnant rats.Adaptive regulation of endothelin receptor type-A and type-B in vascular smooth muscle cells during pregnancy in rats.Epigenetics and developmental programming of adult onset diseases.The microvasculature: a target for nutritional programming and later risk of cardio-metabolic disease.In utero exposure and endometriosis.The impact of early nutrition on the ageing trajectory.Developmental conditioning of the vasculature.The role of maternal nutrition, metabolic function and the placenta in developmental programming of renal dysfunction.KCa3.1 (IK) modulates pancreatic cancer cell migration, invasion and proliferation: anomalous effects on TRAM-34.Nonlinear effects of potassium channel blockers on endothelium-dependent hyperpolarization.Maternal melatonin administration mitigates coronary stiffness and endothelial dysfunction, and improves heart resilience to insult in growth restricted lambs.The management of fibroids based on immunohistochemical studies of their pseudocapsules.Primary Pediatric Hypertension: Current Understanding and Emerging Concepts.Fathers that are born small program alterations in the next-generation preimplantation rat embryos.Cardio-renal and metabolic adaptations during pregnancy in female rats born small: implications for maternal health and second generation fetal growth.Low birth weight followed by postnatal over-nutrition in the guinea pig exposes a predominant player in the development of vascular dysfunction.Maternal alcohol consumption in pregnancy enhances arterial stiffness and alters vasodilator function that varies between vascular beds in fetal sheep.Vascular effects of aerobic exercise training in rat adult offspring exposed to hypoxia-induced intrauterine growth restriction.Uteroplacental insufficiency programmes vascular dysfunction in non-pregnant rats: compensatory adaptations in pregnancy.

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P2860

Uteroplacental insufficiency programs regional vascular dysfunction and alters arterial stiffness in female offspring.

http://www.wikidata.org/entity/Q51570553

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

@zh-my

2010年学术文章

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2010年學術文章

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2010年學術文章

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name

Uteroplacental insufficiency p ...... stiffness in female offspring.

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Uteroplacental insufficiency p ...... stiffness in female offspring.

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The Journal of Physiology

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Uteroplacental insufficiency p ...... stiffness in female offspring

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Helena C Parkington

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Marc Q Mazzuca

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Marianne Tare

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Nicoleta M Dragomir

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P2860

Mechanisms, Pathophysiology, and Therapy of Arterial Stiffness

Dietary restriction in pregnant rats causes gender-related hypertension and vascular dysfunction in offspring

Effect of in utero and early-life conditions on adult health and disease

Impaired acetylcholine-induced vascular relaxation in low birth weight infants: implications for adult hypertension?

Impaired endothelial function and increased carotid stiffness in 9-year-old children with low birthweight.

Impact of low birth weight and cardiovascular risk factors on endothelial function in early adult life.

EDHF, NO and a prostanoid: hyperpolarization-dependent and -independent relaxation in guinea-pig arteries

Endothelial potassium channels, endothelium-dependent hyperpolarization and the regulation of vascular tone in health and disease.

Design and statistical methods in studies using animal models of development.

Physiological and molecular mechanisms governing sexual dimorphism of kidney, cardiac, and vascular function.

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