Effect of inhaled endotoxin on airway and circulating inflammatory cell phagocytosis and CD11b expression in atopic asthmatic subjects.
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CD11c(+)/CD11b(+) cells are critical for organic dust-elicited murine lung inflammationVitamin D supplementation protects against bone loss following inhalant organic dust and lipopolysaccharide exposures in miceRegulation of aeroallergen immunity by the innate immune system: laboratory evidence for a new paradigm.Enhancement of OVA-induced murine lung eosinophilia by co-exposure to contamination levels of LPS in Asian sand dust and heated dust.Allergen tolerance versus the allergic march: the hygiene hypothesis revisited.Atopic asthmatic subjects but not atopic subjects without asthma have enhanced inflammatory response to ozone.Expression of calgranulin A/B heterodimer after acute inhalation of endotoxin: proteomic approach and validation.The glutathione-S-transferase Mu 1 null genotype modulates ozone-induced airway inflammation in human subjects.IL-1 receptor antagonist reduces endotoxin-induced airway inflammation in healthy volunteers.Comparative airway inflammatory response of normal volunteers to ozone and lipopolysaccharide challengeFluticasone propionate protects against ozone-induced airway inflammation and modified immune cell activation markers in healthy volunteers.Alveolar macrophage phagocytosis is impaired in children with poorly controlled asthmaIn vivo gamma-tocopherol supplementation decreases systemic oxidative stress and cytokine responses of human monocytes in normal and asthmatic subjects.Atopic asthmatic patients have reduced airway inflammatory cell recruitment after inhaled endotoxin challenge compared with healthy volunteers.Vitamin E, γ-tocopherol, reduces airway neutrophil recruitment after inhaled endotoxin challenge in rats and in healthy volunteers.Effects of ex vivo γ-tocopherol on airway macrophage function in healthy and mild allergic asthmaticsImmunomodulation by lipid emulsions in pulmonary inflammation: a randomized controlled trial.Inhaled corticosteroid treatment for 6 months was not sufficient to normalize phagocytosis in asthmatic children.Patients with asthma demonstrate airway inflammation after exposure to concentrated ambient particulate matter.Time course of endotoxin-induced airways' inflammation in healthy subjects.Macrophage Phagocytosis and Allergen Avoidance in Children With Asthma
P2860
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P2860
Effect of inhaled endotoxin on airway and circulating inflammatory cell phagocytosis and CD11b expression in atopic asthmatic subjects.
description
2003 nî lūn-bûn
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2003年の論文
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2003年学术文章
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2003年学术文章
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2003年学术文章
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name
Effect of inhaled endotoxin on ...... in atopic asthmatic subjects.
@en
Effect of inhaled endotoxin on ...... in atopic asthmatic subjects.
@nl
type
label
Effect of inhaled endotoxin on ...... in atopic asthmatic subjects.
@en
Effect of inhaled endotoxin on ...... in atopic asthmatic subjects.
@nl
prefLabel
Effect of inhaled endotoxin on ...... in atopic asthmatic subjects.
@en
Effect of inhaled endotoxin on ...... in atopic asthmatic subjects.
@nl
P2093
P921
P356
P1476
Effect of inhaled endotoxin on ...... in atopic asthmatic subjects.
@en
P2093
David B Peden
Marlowe W Eldridge
Neil E Alexis
P304
P356
10.1067/MAI.2003.1651
P407
P577
2003-08-01T00:00:00Z