Cigarette smoke-induced iBALT mediates macrophage activation in a B cell-dependent manner in COPD.
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Application of biclustering of gene expression data and gene set enrichment analysis methods to identify potentially disease causing nanomaterialsTotal particulate matter concentration skews cigarette smoke's gene expression profilePneumocystis-Driven Inducible Bronchus-Associated Lymphoid Tissue Formation Requires Th2 and Th17 ImmunityCoactivator-Associated Arginine Methyltransferase-1 Function in Alveolar Epithelial Senescence and Elastase-Induced Emphysema SusceptibilityHhip haploinsufficiency sensitizes mice to age-related emphysemaTreatment with intranasal iloprost reduces disease manifestations in a murine model of previously established COPDCommunity-Reviewed Biological Network Models for Toxicology and Drug Discovery Applications.Inflammaging increases susceptibility to cigarette smoke-induced COPD.Noncanonical WNT-5A signaling impairs endogenous lung repair in COPDA review of recent developments and applications of morphometry/stereology in lung research.Deep Proteome Profiling Reveals Common Prevalence of MZB1-positive Plasma B Cells in Human Lung and Skin Fibrosis.B cells in chronic obstructive pulmonary disease: moving to center stage.Impairment of Immunoproteasome Function by Cigarette Smoke and in Chronic Obstructive Pulmonary Disease.Il-17A contributes to maintenance of pulmonary homeostasis in a murine model of cigarette smoke-induced emphysema.Application of bi-clustering of gene expression data and gene set enrichment analysis methods to identify potentially disease causing nanomaterials.Cigarette Smoke Regulates the Competitive Interactions between NRF2 and BACH1 for Heme Oxygenase-1 Induction.Reduced Frizzled Receptor 4 Expression Prevents WNT/β-Catenin-driven Alveolar Lung Repair in Chronic Obstructive Pulmonary Disease.Role of B Cell-Activating Factor in Chronic Obstructive Pulmonary Disease.Innate Immunity of the Lung: From Basic Mechanisms to Translational Medicine.Cigarette smoke causes acute airway disease and exacerbates chronic obstructive lung disease in neonatal mice.Cholesterol metabolism promotes B-cell positioning during immune pathogenesis of chronic obstructive pulmonary disease.
P2860
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P2860
Cigarette smoke-induced iBALT mediates macrophage activation in a B cell-dependent manner in COPD.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
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2014年学术文章
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2014年学术文章
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@zh-hans
2014年学术文章
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name
Cigarette smoke-induced iBALT ...... cell-dependent manner in COPD.
@en
Cigarette smoke-induced iBALT ...... cell-dependent manner in COPD.
@nl
type
label
Cigarette smoke-induced iBALT ...... cell-dependent manner in COPD.
@en
Cigarette smoke-induced iBALT ...... cell-dependent manner in COPD.
@nl
prefLabel
Cigarette smoke-induced iBALT ...... cell-dependent manner in COPD.
@en
Cigarette smoke-induced iBALT ...... cell-dependent manner in COPD.
@nl
P2093
P2860
P1476
Cigarette smoke-induced iBALT ...... cell-dependent manner in COPD.
@en
P2093
Ali Önder Yildirim
Gerrit John-Schuster
Johannes Beckers
Katrin Hager
Oliver Eickelberg
P2860
P304
P356
10.1152/AJPLUNG.00092.2014
P577
2014-08-15T00:00:00Z