Eosinophil-epithelial cell interactions: an important facet of asthmatic inflammation.
about
Autophagy mechanisms in sputum and peripheral blood cells of patients with severe asthma: a new therapeutic target.Post-transcriptional silencing of CCR3 downregulates IL-4 stimulated release of eotaxin-3 (CCL26) and other CCR3 ligands in alveolar type II cells.Systematic evaluation of transcellular activities of secretory phospholipases A2. High activity of group V phospholipases A2 to induce eicosanoid biosynthesis in neighboring inflammatory cells.Induction of IL-6 in co-culture of bronchial epithelial cells and eosinophils is regulated by p38 MAPK and NF-kappaB.
P2860
Eosinophil-epithelial cell interactions: an important facet of asthmatic inflammation.
description
2002 nî lūn-bûn
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2002年の論文
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2002年学术文章
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2002年学术文章
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name
Eosinophil-epithelial cell interactions: an important facet of asthmatic inflammation.
@en
Eosinophil-epithelial cell interactions: an important facet of asthmatic inflammation.
@nl
type
label
Eosinophil-epithelial cell interactions: an important facet of asthmatic inflammation.
@en
Eosinophil-epithelial cell interactions: an important facet of asthmatic inflammation.
@nl
prefLabel
Eosinophil-epithelial cell interactions: an important facet of asthmatic inflammation.
@en
Eosinophil-epithelial cell interactions: an important facet of asthmatic inflammation.
@nl
P2860
P1476
Eosinophil-epithelial cell interactions: an important facet of asthmatic inflammation.
@en
P2093
Darren W Sexton
Garry M Walsh
P2860
P304
P356
10.1046/J.1365-2745.2002.01428.X
P577
2002-06-01T00:00:00Z