Investigating the CYP2E1 Potential Role in the Mechanisms Behind INH/LPS-Induced Hepatotoxicity.

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Investigating the CYP2E1 Potential Role in the Mechanisms Behind INH/LPS-Induced Hepatotoxicity.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Investigating the CYP2E1 Poten ...... NH/LPS-Induced Hepatotoxicity.

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Investigating the CYP2E1 Poten ...... NH/LPS-Induced Hepatotoxicity.

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label

Investigating the CYP2E1 Poten ...... NH/LPS-Induced Hepatotoxicity.

@en

Investigating the CYP2E1 Poten ...... NH/LPS-Induced Hepatotoxicity.

@nl

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Investigating the CYP2E1 Poten ...... NH/LPS-Induced Hepatotoxicity.

@en

Investigating the CYP2E1 Poten ...... NH/LPS-Induced Hepatotoxicity.

@nl

P1476

Investigating the CYP2E1 Poten ...... NH/LPS-Induced Hepatotoxicity.

@en

P2093

Hongli Guo

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Hozeifa M Hassan

http://www.w3.org/2001/XMLSchema#string

Liu Xiaoxin

http://www.w3.org/2001/XMLSchema#string

Luyong Zhang

http://www.w3.org/2001/XMLSchema#string

Zhenzhou Jiang

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CYP2E1 potentiation of LPS and TNFα-induced hepatotoxicity by mechanisms involving enhanced oxidative and nitrosative stress, activation of MAP kinases, and mitochondrial dysfunction.

Mechanisms of inhibition of chemical toxicity and carcinogenesis by diallyl sulfide (DAS) and related compounds from garlic.

Bile acid signaling and liver regeneration.

Toxic hepatitis with isoniazid and rifampin. A meta-analysis.

Endothelium-derived Toll-like receptor-4 is the key molecule in LPS-induced neutrophil sequestration into lungs.

Amiodarone exposure during modest inflammation induces idiosyncrasy-like liver injury in rats: role of tumor necrosis factor-alpha.

Interactions between lipopolysaccharide and the intestinal epithelium.

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198

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scholarly article

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10.3389/FPHAR.2018.00198

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P478

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P50

Bashir A Yousef

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2018-03-07T00:00:00Z

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P698

29563874

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P921

hepatotoxicity

P932

5850051

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