Bone mineral density in Klinefelter syndrome is reduced and primarily determined by muscle strength and resorptive markers, but not directly by testosterone.
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Recognizing and treating secondary osteoporosisThe Sex Chromosome Trisomy mouse model of XXY and XYY: metabolism and motor performance.Criminality in men with Klinefelter's syndrome and XYY syndrome: a cohort study.Primary hyperparathyroidism and Klinefelter's syndrome in a young man.Aberrant gene expression profiles in pluripotent stem cells induced from fibroblasts of a Klinefelter syndrome patient.Bone Mineral Status in Children and Adolescents with Klinefelter Syndrome.The macrophage low-grade inflammation marker sCD163 is modulated by exogenous sex steroids.Klinefelter syndrome (KS): genetics, clinical phenotype and hypogonadism.Progressive Improvement of T-Scores in Men with Osteoporosis and Subnormal Serum Testosterone Levels upon Treatment with Testosterone over Six Years.The role of hypogonadism in Klinefelter syndrome.Male Hypogonadism and Osteoporosis: The Effects, Clinical Consequences, and Treatment of Testosterone Deficiency in Bone Health.47,XXY Klinefelter syndrome: clinical characteristics and age-specific recommendations for medical management.Klinefelter syndrome: the commonest form of hypogonadism, but often overlooked or untreated.Cell-free DNA screening and sex chromosome aneuploidies.Role of vitamin D levels and vitamin D supplementation on bone mineral density in Klinefelter syndrome.Short QTc interval in males with klinefelter syndrome-influence of CAG repeat length, body composition, and testosterone replacement therapy.Low INSL3 in Klinefelter syndrome is related to osteocalcin, testosterone treatment and body composition, as well as measures of the hypothalamic-pituitary-gonadal axis.Bone geometry, volumetric density, microarchitecture, and estimated bone strength assessed by HR-pQCT in Klinefelter syndrome.Genotype and phenotype in Klinefelter syndrome - impact of androgen receptor polymorphism and skewed X inactivationDNA hypermethylation and differential gene expression associated with Klinefelter syndrome
P2860
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P2860
Bone mineral density in Klinefelter syndrome is reduced and primarily determined by muscle strength and resorptive markers, but not directly by testosterone.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh-hant
name
Bone mineral density in Klinef ...... not directly by testosterone.
@en
Bone mineral density in Klinef ...... not directly by testosterone.
@nl
type
label
Bone mineral density in Klinef ...... not directly by testosterone.
@en
Bone mineral density in Klinef ...... not directly by testosterone.
@nl
prefLabel
Bone mineral density in Klinef ...... not directly by testosterone.
@en
Bone mineral density in Klinef ...... not directly by testosterone.
@nl
P2093
P2860
P1476
Bone mineral density in Klinef ...... not directly by testosterone.
@en
P2093
J S Christiansen
K Kristensen
L Heickendorff
L Mosekilde
N Birkebæk
P2860
P2888
P304
P356
10.1007/S00198-010-1354-7
P407
P577
2010-07-24T00:00:00Z