TGF β-1 administration during ex vivo expansion of human articular chondrocytes in a serum-free medium redirects the cell phenotype toward hypertrophy.
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The Regulatory Role of Signaling Crosstalk in Hypertrophy of MSCs and Human Articular ChondrocytesCytokine networking of chondrocyte dedifferentiation in vitro and its implications for cell-based cartilage therapyAIMP1 downregulation restores chondrogenic characteristics of dedifferentiated/degenerated chondrocytes by enhancing TGF-β signal.The Good the Bad and the Ugly of Glycosaminoglycans in Tissue Engineering ApplicationsGeneration of Col2a1-EGFP iPS cells for monitoring chondrogenic differentiation.Transforming growth factor beta signaling is essential for the autonomous formation of cartilage-like tissue by expanded chondrocytesStrategies to minimize hypertrophy in cartilage engineering and regeneration.Chondrogenic differentiation of amniotic fluid stem cells and their potential for regenerative therapy.Recapitulating endochondral ossification: a promising route to in vivo bone regeneration.S100B + A1 CELISA: A Novel Potency Assay and Screening Tool for Redifferentiation Stimuli of Human Articular Chondrocytes.Silencing of anti-chondrogenic microRNA-221 in human mesenchymal stem cells promotes cartilage repair in vivo.No effect of subperiosteal growth factor application on periosteal neo-chondrogenesis in osteoperiosteal bone grafts for osteochondral defect repair.Serum- and growth-factor-free three-dimensional culture system supports cartilage tissue formation by promoting collagen synthesis via Sox9-Col2a1 interaction.Control of collagen production in mouse chondrocytes by using a combination of bone morphogenetic protein-2 and small interfering RNA targeting Col1a1 for hydrogel-based tissue-engineered cartilage.Protection of bovine chondrocyte phenotype by heat inactivation of allogeneic serum in monolayer expansion cultures.Human aqueous humor levels of transforming growth factor-β2: Association with matrix metalloproteinases/tissue inhibitors of matrix metalloproteinases.TGFβ1 induces hypertrophic change and expression of angiogenic factors in human chondrocytes.Exposure to reversine affects the chondrocyte morphology and phenotype in vitro.In Vitro Analysis of the Differentiation Capacity of Postmortally Isolated Human Chondrocytes Influenced by Different Growth Factors and Oxygen Levels.Chondrogenesis by bone marrow-derived mesenchymal stem cells grown in chondrocyte-conditioned medium for auricular reconstruction.
P2860
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P2860
TGF β-1 administration during ex vivo expansion of human articular chondrocytes in a serum-free medium redirects the cell phenotype toward hypertrophy.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
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2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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name
TGF β-1 administration during ...... phenotype toward hypertrophy.
@en
TGF β-1 administration during ...... phenotype toward hypertrophy.
@nl
type
label
TGF β-1 administration during ...... phenotype toward hypertrophy.
@en
TGF β-1 administration during ...... phenotype toward hypertrophy.
@nl
prefLabel
TGF β-1 administration during ...... phenotype toward hypertrophy.
@en
TGF β-1 administration during ...... phenotype toward hypertrophy.
@nl
P2093
P2860
P356
P1476
TGF β-1 administration during ...... phenotype toward hypertrophy.
@en
P2093
P2860
P304
P356
10.1002/JCP.24024
P577
2012-09-01T00:00:00Z