Phosphorylation of Akt (Ser473) is an excellent predictor of poor clinical outcome in prostate cancer. - Wikidata - wikimedia - TriplyDB

Phosphorylation of Akt (Ser473) is an excellent predictor of poor clinical outcome in prostate cancer.

Phosphorylation of Akt (Ser473) is an excellent predictor of poor clinical outcome in prostate cancer.

http://www.wikidata.org/entity/Q51992670

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Identification of a novel inhibitor of differentiation-1 (ID-1) binding partner, caveolin-1, and its role in epithelial-mesenchymal transition and resistance to apoptosis in prostate cancer cells Protein kinases and phosphatases in the control of cell fate The importance of non-nuclear AR signaling in prostate cancer progression and therapeutic resistance Targeted therapy for advanced prostate cancer: inhibition of the PI3K/Akt/mTOR pathway Active monitoring of formaldehyde diffusion into histological tissues with digital acoustic interferometry.Cyclin-dependent kinase 5 acts as a critical determinant of AKT-dependent proliferation and regulates differential gene expression by the androgen receptor in prostate cancer cells Sphingosine kinase-1 is central to androgen-regulated prostate cancer growth and survival.Upregulation of PTEN involved in rosiglitazone-induced apoptosis in human hepatocellular carcinoma cells.MP470, a novel receptor tyrosine kinase inhibitor, in combination with Erlotinib inhibits the HER family/PI3K/Akt pathway and tumor growth in prostate cancer Proteomics identification of desmin as a potential oncofetal diagnostic and prognostic biomarker in colorectal cancer.Association of the actin-binding protein transgelin with lymph node metastasis in human colorectal cancer Comparison of Akt/mTOR signaling in primary breast tumors and matched distant metastases Inositol hexaphosphate suppresses growth and induces apoptosis in prostate carcinoma cells in culture and nude mouse xenograft: PI3K-Akt pathway as potential target.1,4-phenylenebis(methylene)selenocyanate, but not selenomethionine, inhibits androgen receptor and Akt signaling in human prostate cancer cells.Comparative proteomic approach identifies PKM2 and cofilin-1 as potential diagnostic, prognostic and therapeutic targets for pulmonary adenocarcinoma Impact of circulating cholesterol levels on growth and intratumoral androgen concentration of prostate tumors Caffeic acid phenethyl ester causes p21 induction, Akt signaling reduction, and growth inhibition in PC-3 human prostate cancer cells.A computational model to predict bone metastasis in breast cancer by integrating the dysregulated pathways.Akt activation is a common event in pediatric malignant gliomas and a potential adverse prognostic marker: a report from the Children's Oncology Group.Synergistic simvastatin and metformin combination chemotherapy for osseous metastatic castration-resistant prostate cancer.Genetic variations in a PTEN/AKT/mTOR axis and prostate cancer risk in a Chinese population.Clinical application for the preservation of phospho-proteins through in-situ tissue stabilization Predictive value of Sp1/Sp3/FLIP signature for prostate cancer recurrence.Phospho-Akt immunoreactivity in prostate cancer: relationship to disease severity and outcome, Ki67 and phosphorylated EGFR expression Receptor-recognized α₂-macroglobulin binds to cell surface-associated GRP78 and activates mTORC1 and mTORC2 signaling in prostate cancer cells.Association between cannabinoid CB₁ receptor expression and Akt signalling in prostate cancer.Cyclic phosphatidic acid stimulates cAMP production and inhibits growth in human colon cancer cells.Difference in protein expression profile and chemotherapy drugs response of different progression stages of LNCaP sublines and other human prostate cancer cells Castration-resistant prostate cancer: new science and therapeutic prospects.Androgen receptor signaling in prostate cancer development and progression.Development of a peptide-drug conjugate for prostate cancer therapy.Transcription of Nrdp1 by the androgen receptor is regulated by nuclear filamin A in prostate cancer Immunohistochemical Assessment of Phosphorylated mTORC1-Pathway Proteins in Human Brain Tumors.Caffeic acid phenethyl ester induced cell cycle arrest and growth inhibition in androgen-independent prostate cancer cells via regulation of Skp2, p53, p21Cip1 and p27Kip1.Antagonists of growth hormone-releasing hormone inhibit growth of androgen-independent prostate cancer through inactivation of ERK and Akt kinases Expression of phosphorylated Akt/mTOR and clinical significance in human ameloblastoma.AGE/RAGE/Akt pathway contributes to prostate cancer cell proliferation by promoting Rb phosphorylation and degradation The CXCL12/CXCR4 axis promotes ligand-independent activation of the androgen receptor Abrogation of de novo lipogenesis by stearoyl-CoA desaturase 1 inhibition interferes with oncogenic signaling and blocks prostate cancer progression in mice.EGF prevents the neuroendocrine differentiation of LNCaP cells induced by serum deprivation: the modulator role of PI3K/Akt

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P2860

Phosphorylation of Akt (Ser473) is an excellent predictor of poor clinical outcome in prostate cancer.

http://www.wikidata.org/entity/Q51992670

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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name

Phosphorylation of Akt (Ser473 ...... al outcome in prostate cancer.

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Phosphorylation of Akt

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Phosphorylation of Akt (Ser473 ...... al outcome in prostate cancer.

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Phosphorylation of Akt

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Phosphorylation of Akt (Ser473 ...... al outcome in prostate cancer.

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Phosphorylation of Akt

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Cancer Research

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Phosphorylation of Akt (Ser473 ...... al outcome in prostate cancer.

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Dean A Troyer

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Jeffrey I Kreisberg

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Paramita M Ghosh

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Roble G Bedolla

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Shazli N Malik

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Suzanne Kreisberg

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Thomas J Prihoda

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5232-5236

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scholarly article

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