Potassium inhibition of sodium-activated potassium (K(Na)) channels in guinea-pig ventricular myocytes.
about
Knockout of Slo2.2 enhances itch, abolishes KNa current, and increases action potential firing frequency in DRG neurons.The N-terminal domain of Slack determines the formation and trafficking of Slick/Slack heteromeric sodium-activated potassium channels.Characterization and influence of cardiac background sodium current in the atrioventricular node.THE ROLE OF INTRACELLULAR SODIUM (Na) IN THE REGULATION OF CALCIUM (Ca)-MEDIATED SIGNALING AND TOXICITY.Activation of Slo2.1 channels by niflumic acid.Na+-activated K+ current contributes to postexcitatory hyperpolarization in neocortical intrinsically bursting neurons.
P2860
Potassium inhibition of sodium-activated potassium (K(Na)) channels in guinea-pig ventricular myocytes.
description
2000 nî lūn-bûn
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name
Potassium inhibition of sodium ...... inea-pig ventricular myocytes.
@en
Potassium inhibition of sodium-activated potassium
@nl
type
label
Potassium inhibition of sodium ...... inea-pig ventricular myocytes.
@en
Potassium inhibition of sodium-activated potassium
@nl
prefLabel
Potassium inhibition of sodium ...... inea-pig ventricular myocytes.
@en
Potassium inhibition of sodium-activated potassium
@nl
P2860
P1476
Potassium inhibition of sodium ...... inea-pig ventricular myocytes.
@en
P2093
P2860
P356
10.1111/J.1469-7793.2000.00081.X
P407
P478
P577
2000-07-01T00:00:00Z