Antioxidants diminish developmental damage induced by high glucose and cyclooxygenase inhibitors in rat embryos in vitro.
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Altered gene expression in rat cranial neural crest cells exposed to a teratogenic glucose concentration in vitro: paradoxical downregulation of antioxidative defense genesDecreased glucose transporter expression triggers BAX-dependent apoptosis in the murine blastocystImmunohistochemical identification and quantitative analysis of cytoplasmic Cu/Zn superoxide dismutase in mouse organogenesisPathogenesis of diabetes-induced congenital malformations.Impact of retinoic acid exposure on midfacial shape variation and manifestation of holoprosencephaly in Twsg1 mutant mice.Free radicals and birth defects.Advances in understanding the molecular causes of diabetes-induced birth defects.Experimental mechanisms of diabetic embryopathy and strategies for developing therapeutic interventions.Frontiers in research on maternal diabetes-induced neural tube defects: Past, present and futureMaternal hyperglycemia activates an ASK1-FoxO3a-caspase 8 pathway that leads to embryonic neural tube defectsNew concepts in diabetic embryopathyThe status of diabetic embryopathy.Diabetes and apoptosis: neural crest cells and neural tube.Acetylsalicylic acid interferes with embryonic kidney growth and development by a prostaglandin-independent mechanism.Effect of maternal diabetes on the embryo, fetus, and children: congenital anomalies, genetic and epigenetic changes and developmental outcomes.The fructosamine 3-kinase knockout mouse: a tool for testing the glycation hypothesis of intracellular protein damage in diabetes and aging.Metabolite availability as a window to view the early embryo microenvironment in vivo.Induction of embryonic dysmorphogenesis by high glucose concentration, disturbed inositol metabolism, and inhibited protein kinase C activity.Oxidant regulation of gene expression and neural tube development: Insights gained from diabetic pregnancy on molecular causes of neural tube defects.Dietary supplementation with Ipomoea aquatica (whole leaf powder) attenuates maternal and fetal oxidative stress in streptozotocin-diabetic rats.Melatonin prevents neural tube defects in the offspring of diabetic pregnancy.Role of reactive oxygen species in gynecologic diseases.
P2860
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P2860
Antioxidants diminish developmental damage induced by high glucose and cyclooxygenase inhibitors in rat embryos in vitro.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Antioxidants diminish developm ...... itors in rat embryos in vitro.
@en
Antioxidants diminish developm ...... itors in rat embryos in vitro.
@nl
type
label
Antioxidants diminish developm ...... itors in rat embryos in vitro.
@en
Antioxidants diminish developm ...... itors in rat embryos in vitro.
@nl
prefLabel
Antioxidants diminish developm ...... itors in rat embryos in vitro.
@en
Antioxidants diminish developm ...... itors in rat embryos in vitro.
@nl
P1433
P1476
Antioxidants diminish developm ...... itors in rat embryos in vitro.
@en
P2093
Eriksson UJ
P304
P356
10.2337/DIABETES.47.4.677
P407
P577
1998-04-01T00:00:00Z