Hyperexcitability in a model of cortical maldevelopment.
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Brain weight differences associated with induced focal microgyria.Temporal and topographic alterations in expression of the alpha3 isoform of Na+, K(+)-ATPase in the rat freeze lesion model of microgyria and epileptogenesisHistometric changes and cell death in the thalamus after neonatal neocortical injury in the rat.Enhanced synaptic connectivity and epilepsy in C1q knockout mice.Dampened hippocampal oscillations and enhanced spindle activity in an asymptomatic model of developmental cortical malformationsContinuous spike-waves during slow-wave sleep in a mouse model of focal cortical dysplasiaExcitatory and inhibitory synaptic connectivity to layer V fast-spiking interneurons in the freeze lesion model of cortical microgyria.Electrophysiological and morphological characterization of neurons within neocortical ectopias.Selective alterations in glutamate and GABA receptor subunit mRNA expression in dysplastic neurons and giant cells of cortical tubers.Enhanced infragranular and supragranular synaptic input onto layer 5 pyramidal neurons in a rat model of cortical dysplasia.HCN Channel Modulation of Synaptic Integration in GABAergic Interneurons in Malformed Rat Neocortex.Upregulation of glutamate receptors in rat cerebral cortex with neuronal migration disordersIncreased pyramidal excitability and NMDA conductance can explain posttraumatic epileptogenesis without disinhibition: a model.Abnormal pyramidal cell morphology and HCN channel expression in cortical dysplasia.Cellular abnormalities and synaptic plasticity in seizure disorders of the immature nervous system.Cortical malformations and epilepsy.Gabapentin attenuates hyperexcitability in the freeze-lesion model of developmental cortical malformation.Blood-brain barrier breakdown following traumatic brain injury: a possible role in posttraumatic epilepsy.Functional changes in glutamate transporters and astrocyte biophysical properties in a rodent model of focal cortical dysplasiaCortical dysgenesis and epilepsy.Homeostatic increase in excitability in area CA1 after Schaffer collateral transection in vivo.Laser-scanning astrocyte mapping reveals increased glutamate-responsive domain size and disrupted maturation of glutamate uptake following neonatal cortical freeze-lesion.Decreased hyperpolarization-activated currents in layer 5 pyramidal neurons enhances excitability in focal cortical dysplasia.Initiation of epileptiform activity in a rat model of periventricular nodular heterotopiaAre Ectopic Neurons a Red Herring in Localizing Seizure Foci?REORGANIZATION OF BARREL CIRCUITS LEADS TO THALAMICALLY-EVOKED CORTICAL EPILEPTIFORM ACTIVITY.Glutamate biosensor imaging reveals dysregulation of glutamatergic pathways in a model of developmental cortical malformationAstrocyte membrane properties are altered in a rat model of developmental cortical malformation but single-cell astrocytic glutamate uptake is robust.Pathophysiological mechanisms of focal cortical dysplasia: a critical review of human tissue studies and animal models.Decreased glutamate transport enhances excitability in a rat model of cortical dysplasiaDcx reexpression reduces subcortical band heterotopia and seizure threshold in an animal model of neuronal migration disorderEnhanced Burst-Suppression and Disruption of Local Field Potential Synchrony in a Mouse Model of Focal Cortical Dysplasia Exhibiting Spike-Wave Seizures.Blood-brain barrier breakdown-inducing astrocytic transformation: novel targets for the prevention of epilepsy.Early susceptibility for epileptiform activity in malformed cortexAnimal models of focal cortical dysplasia and tuberous sclerosis complex: recent progress toward clinical applications.Accumulation of GABAergic neurons, causing a focal ambient GABA gradient, and downregulation of KCC2 are induced during microgyrus formation in a mouse model of polymicrogyria.Epileptogenesis in the Dysplastic Brain: A Revival of Familiar ThemesPotentially epileptogenic dysfunction of cortical NMDA- and GABA-mediated neurotransmission in Otx1-/- mice.Endogenous subventricular zone neural progenitors contribute to the formation and hyperexcitability of experimental model of focal microgyria.Electrophysiological characteristics of reactive astrocytes in experimental cortical dysplasia.
P2860
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P2860
Hyperexcitability in a model of cortical maldevelopment.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh-hant
name
Hyperexcitability in a model of cortical maldevelopment.
@en
Hyperexcitability in a model of cortical maldevelopment.
@nl
type
label
Hyperexcitability in a model of cortical maldevelopment.
@en
Hyperexcitability in a model of cortical maldevelopment.
@nl
prefLabel
Hyperexcitability in a model of cortical maldevelopment.
@en
Hyperexcitability in a model of cortical maldevelopment.
@nl
P356
P1433
P1476
Hyperexcitability in a model of cortical maldevelopment.
@en
P2093
P304
P356
10.1093/CERCOR/6.3.514
P577
1996-05-01T00:00:00Z