Cholecystokinin decreases sucrose intake in chronic decerebrate rats.
about
Neuroendocrine regulation of appetitive ingestive behaviorLeptin and the systems neuroscience of meal size control.Neurobiology of inflammation-associated anorexia.Adrenoreceptor modulation of oromotor pathways in the rat medullaAscending projections from the caudal visceral nucleus of the solitary tract to brain regions involved in food intake and energy expenditure.CCK inhibits the orexigenic effect of peripheral ghrelin.A novel slice preparation to study medullary oromotor and autonomic circuits in vitro.Pharmacological treatment of obesity in children and adolescents: present and futureMelanocortin control of energy balance: evidence from rodent models.Gastrointestinal regulation of food intake.Clinical review: Regulation of food intake, energy balance, and body fat mass: implications for the pathogenesis and treatment of obesityActivation of NPY receptors suppresses excitatory synaptic transmission in a taste-feeding network in the lower brain stem.The taste of sugars.Visceral sensory inputs to the endocrine hypothalamus.Hindbrain glucagon-like peptide-1 neurons track intake volume and contribute to injection stress-induced hypophagia in meal-entrained ratsCentral control of body weight and appetite.2-Deoxy-D-glucose, but not mercaptoacetate, increases food intake in decerebrate rats.Pathological satiety caused by brainstem hemangioblastoma.Immortalized neurons for the study of hypothalamic function.Gastric stimulation for weight loss.Hindbrain neurons as an essential hub in the neuroanatomically distributed control of energy balance.Interoceptive modulation of neuroendocrine, emotional, and hypophagic responses to stress.Overnight food deprivation markedly attenuates hindbrain noradrenergic, glucagon-like peptide-1, and hypothalamic neural responses to exogenous cholecystokinin in male rats.Differential activation of chemically identified neurons in the caudal nucleus of the solitary tract in non-entrained rats after intake of satiating vs. non-satiating meals.Satiation and stress-induced hypophagia: examining the role of hindbrain neurons expressing prolactin-releasing Peptide or glucagon-like Peptide 1.Loss of cholecystokinin and glucagon-like peptide-1-induced satiation in mice lacking serotonin 2C receptors.
P2860
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P2860
Cholecystokinin decreases sucrose intake in chronic decerebrate rats.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年学术文章
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1988年学术文章
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1988年学术文章
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1988年学术文章
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1988年学术文章
@zh-my
1988年学术文章
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1988年學術文章
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1988年學術文章
@zh-hant
name
Cholecystokinin decreases sucrose intake in chronic decerebrate rats.
@en
Cholecystokinin decreases sucrose intake in chronic decerebrate rats.
@nl
type
label
Cholecystokinin decreases sucrose intake in chronic decerebrate rats.
@en
Cholecystokinin decreases sucrose intake in chronic decerebrate rats.
@nl
prefLabel
Cholecystokinin decreases sucrose intake in chronic decerebrate rats.
@en
Cholecystokinin decreases sucrose intake in chronic decerebrate rats.
@nl
P1476
Cholecystokinin decreases sucrose intake in chronic decerebrate rats.
@en
P2093
P304
P356
10.1152/AJPREGU.1988.254.6.R853
P407
P433
P577
1988-06-01T00:00:00Z